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Comments ...

 +0  (nbme24#8)
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I htthugo fo it as, a aeBt aotigns duowl sceua nldot,iaasivo adn eefxlr ca,drythacai reh lpsue is relayda ta 100, os ti odluw jpum hhire.g heS coudl teg otiptaipasnl dna sroe.trm yorbablP the talse ieykll anxpitlnoea uto fo the sert hhgot.u


 +3  (nbme23#48)
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I ujts zerleaid atht hte seawnr I eeelcdst oeludvw' eben nceedrodis pvssaie g:viegsrsea Hvae" yuo densorcedi tnalkgi to a ithfa lheaer to ees if ereth aer yan oreht ssmureae uoy osldhu t"r?y

loam


 +9  (nbme23#16)
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iEtslxnonapa ofr tshi rea too t.decialcmop Tihkn fo ti kiel htis:

oev'uY tog a eipce of uedattm AND that si blea ot eb isgeddet by a srnttoceiir onl,aneceeusd hatt enasm eth AND aws sntipcranoairltyl elaliavba ot nebgi twi.h KAA ti saw ton lmet,hyeatd easubec sa ew nkow, talthiyemno = hitaotmreoenhrc whihc is tnalsaprntolrcyii iictvane. ahtt asnme hyetsleam asw tetmdau

lnOy reoht psllbiuae sarwen aws N,Desa nda if it aws muatdet ti wdoul be iteanvic, ton vtevciraoe.

djeffs1  I mean sure... but this is a prokaryote... +

 +26  (nbme20#20)
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oYu anc rsosc uot hte otp reeht awrsne choiec,s C.,B,A You tonw be obrnbsrgaie thnayign in eht PTC ni sif'nonac mo.nyedsr onogkiL ta lmpioeh,akay athimor,eanyp and oapptyoheipahmhs o.wn lHpeakoamyi 'antc be rccorte ueseacb veen htghou aoptmsius si tlos it liwl be obrdbrseea ta hte aletr ichkt naidcgsen oolp and fi thta osntde emka ,esnse eth ybod liwl judsta ofr low uemrs pitmouass tub ngcativiat hte ++K/H ppmu on lcle.s It tsi'n aiethmrpyoan euscabe ta eth ocigllcetn utdc lpicrpnai llsec, ptrbioroenas wlli .ucorc This eeavls ahotpmsiyeohphpa sa eth ctocrer nad yoln awresn iheocc.

imgdoc  by* +2
larascon  Excellent explanation, thank you ! +1
waterloo  It's worth mentioning, that 25% of Na is reabs in the thicc ascending limb. 67% of filtered Na is reabsorbed in the PCT, whereas 85% of phosphate reabsorbed in the PCT. So pt more likely to show Hypophos. +
coconut  UWorld QID:7626 says lab results in people with Fanconi syndrome will show hypokalemia +
j44n  coconut i thought that too but then i remebred fanconi is associated with hypophosphatemic rickets/ VIT D resistant rickets and this little dude is going to be growing because he's 5 and thats gonna increase the demand for phosphate in his body +

 +1  (nbme20#43)
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I nkow is't niptgtme ot hnikt that shti aym eb geonndrA iteiiyisvnnts somrdeny utb sit t,on rfo ti to eb IAS hteer ndeede ot eb an eancrsei in en.rstttseooe In iths ecsa it swa in het rlmnao neagr rof a feema.l oNw ahtt ew onkw het eonrps si ineiflyted lameef -;&tg Mnrluaile sasneegi eomecbs the ebst nwesr.a


 +18  (nbme21#33)
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I gtutohh taobu it liek :thsi itaePnt si hagivn oenhygiscpc ED - sti all in his head, bldioi si mnlaro dan unrnaclot eensiotcr ear mlr.nao

fI shi rteTosotesne vleel aws owl - Ldbiio ce.arddsee If his voeusnr estmsy tpcitha/ac(impmetets)yayshrap wsa dgdaema - nlnacortu crsetnioe d.raecesde

eopH hist lephs.

sars  Idk about this. My thoughts: -libido is a product of testosterone and emotions (psychogenic) -tumescence is a product of pelvic splanchnics S2-S4 (parasympathetic) -patient's issues are 1 month old (aka recent) and his wife died 2 years ago & testosterone is normal (therefore libido normal) -patient can jack off, therefore penile stimulation reflex arc is intact and pelvic splanchnics are working (nocturnal tumescence normal) +2

 +6  (nbme21#12)
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Teh lclicnia nenrietosatp si htat of lewor anidolbam pani, v,eerf nda ich.lls shTi eolna edam me ihntk it saw an ofaimlmynrat speorc.s lAos eht tieuonqs ysas tereh aer 3 speaetar rpoloy mliideted insrgoe fo arornw nmlu.e As arf as viecerautl cloitsi si noder,ecnc tehre era on pksi isosl,ne ti si onnouisuct reehwvre it is. shTi cudpole iwht eht rthiosy of otipctnnoias smkae tiutidvirlseci the btse wranes oe.ihcc

lovebug  I know it's silly Q. but why not C) Granulomatous colitis? lol. thank you! +1
trazobone  Granulomatous colitis is Chron’s, which also doesn’t fit bc it presents commonly in the terminal ileum and does not usually present with fever as it is an inflammatory pathology rather an infectious one. While it does present with string sign (“narrowing of the lumen”), anything can cause a narrowing of the lumen, so this doesn’t necessarily mean that it’s string sign +1

 +0  (nbme21#15)
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elcIociol is(to/rmeynicrs)eif estcrmneie pr,o)t(al dna nielscp p)(tearllfto/ aelrn ce(siytm)s are het ynlo two rismsptotyeoc htsun osi.tpon fO hicwh cpniels dna eflt alnre is eht SETB toonip fo teh wto stuj eadbs on htire tiximrpoy to eht eaoesglhap cav.iser

msyrett  ileocolic vein is a portal vein, it empties into the Superior Mesenteric Vein +1

 +2  (nbme21#33)
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inoQtseu rtnpssee an urusosppmdemnies ptianet htwi hsar orev eth rewlo akcb hcihw is curtiipr. Vsscieel mas(ll diful ldefli )tbsseilr ear ertnpse twhi crutdesceetr/laud eilsosn sloa undof ni thta arae. sTih is railclvea zosret coif:nteni mtiuelpl rcosp fo slnieso ni uoiarsv eatsgs mofr isvecels to uss.tcr


 +7  (nbme21#4)
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shiT tuiqeons si gksain botua het nuslnii etorprce tsoyenri kinsae taawpy.h So aket it fomr e,reht

  1. eTh paisoyhnhrolpto of the RSI aettivsac a gislna rdtatnuisnoc secdaac tath ealsd to eth inotaatvic of oreth kseasin as wlel as tstnrapiinroc sfrotac htat eemtaid teh anctirlrlauel ecsfetf of uilni.sn tCacepauocsrlN/iylm slhigntut - sye rr(livesbee)

  2. eTnheerni/nSiore aenikss era losa kownn ot deecur hte ycvtitia fo lniui.sn - sye rriseeble)v(

  3. tiuinUqbi - aiddtmee osisyerptol - no not( lebs,rievre) dan losa iinnlsu ambioytllaecl eeincssra ntroepi yhistness os it osdt'en mhcta wtha iuslinn sdeo w.ysnyaa

coertrc me if Im' onwrg.

usmle11a  i think youre correct cause ubiquitin mediated proteolyis is an irreversible step. +1

 +0  (nbme21#48)
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I ntkih hte swearn was ncnouesss sabceue eht uoga""rersst aak lifaym rmesebm anehvt adtidelbere ty.e fI siht swa efart tehir aidtrinobeel dan yteh rgeas,dei nhte ti goes uosesp -;g&t latdu nrecilhd &t;g- tpanres g&-t; siibslng .cet eTh iqenotsu toe'sdn ineonmt ayn emgsdeitera,ns neehc yeht ened ot edidec ctuvlulaiemy adn akme a sieoc.ind

That saw ujst ym eatk on this si.euotqn


 +6  (nbme21#11)
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cilsyBaal shti qutsneoi ujst sska ahtw" si rpe"wo nda ssak uyo to anxleip ttah %80 poer.w woerP si 1 - eatb etp(y 2 er)r,ro ylasabcil henw a cdeeeiffrn issext adn eth nllu heyipssoth is eceertdj si ewp.ro So fi hte Rx tdctsee a maen neercdfife of 40. ni hmtsaa in het naseitpt ni eth aetmettnr r,pugo hnet htat adta lfasl ni teh 8%0 wepor rnae,g dna sti igceniisanfc si %95 0),p0;&t5(.l P lavue is just het orbitpalyib of shonigemt peahngpin by nehcc,a so yuo what this ot be lsse atth 5% so rtawvehe uoy rosbdeve t'sin hluibtsl.

I peho isht hedlp,e nda crtcroe em fi Im' .gnwro

yssya1992  I have a question : whats the relation then between power and P value ? +
privatejoker  This one took me a minute and was during the last block so my brain was already fried. But my reasoning was that, as stated above, since it gave you power, it is basically just a long-winded way of asking what Power is, and how this relates to p-value. P-value is the odds that the finding was due to chance alone. Obviously a p-value set to <0.05 implies a greater than 95% chance that the finding is legit. Since the power is said to be 80%, this means that there is an 80% chance that the study finding is legit, at least insomuch that it met the pre-set criteria of being 95% non-chance related. +
sahusema  80% chance (power) the study correctly identifies the existence of an association in reality. If an association is determined to exist, >95% chance the study and reality agree with each other (p<.05) +1

 +27  (nbme22#37)
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I kinht sti the rhtgi LMF r(eaa ),C not het gihtr buncades nulcuse tath is ienslode no the scosr o.nseict If het eucsdnab eslucun rewe nliesdeo ehs 'onltduw vhae bioucnatd in ehr etfl y.ee eTh FLM etaidems ssroc tlka ebtnewe eth cunbeads seculun no htob sidse to teh FLM no teh potepois eids 2( cbnuasde ceiu,ln 2 MFL noe no ahce ds)e.i In ehr easc, erh grhti LFM s'twna ictinognfun enche ywh hes asw zgngai felt tub erh htrgi edlmid utcrse astwn' ignnccaott to tdimaee ewlartdf e.gaz

isTh rpetcui sh:pel ow0ohtw/5_a9c041dtes.p2/--nFtwt-t/ommp1hg2//ut/:tpcpbhnmhhw.peojnlua/

imgdoc  *medial rectus +

 +20  (nbme22#1)
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I ihktn tlao of elppeo mgthi hvea roev mpeszaedih woh iamnttorp NAP adn PNB rllyea aer, eys ti is tptarnmoi to nkwo teesh sietdppe gte ertecsed by eth vintreaarlrl/ucita aorimucmdy ugdnri etrha .rlfuaie Hvwroee retih lveoalr enifsvesfceet in taergint trahe fuailre is l,izhc a eceortppr tdol me atth fi NPA nad PNB ewer so usfule in enrtiruiass tenh ywh do we iveg ierdstc?ui sIt' uceesba SARA oersoevwrp isth emtyss chnee anicgsu vteaneig esefftc dan hte eselnds polo fo htrae le.iafru KAA ywh ew iegv ECA b.itsinrhoi

niogKnw htta ANP setg znuiadeetlr by eth SAAR m,syste ew anc ishtf uro uofsc kacb to arteh lefaiur in hsit apntite, ewreh airdacc puoutt si e,asdeercd ldeanig to AHD ctnseoier dan liyanfl nldoiiulat p.yotnmhareia

almondbreeze  a concept continuously emphasized by uw, but I get always wrong :'( +2
almondbreeze  good work done! +
raffff  why does the body make anp at all since its so useless +3
makinallkindzofgainz  @raffff - at least BNP gives us a good marker for heart failure exacerbations :) thanks body! +
mannan  Yeah it's important clinically because it has a high sensitivity (if negative, rule out) for Heart failure. +
alimd  At the same time ANP inhibits renin release? +




Subcomments ...

submitted by imgdoc(135),
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uYo acn ocssr uot het opt erthe sanwer h,iecocs ,.,ABC You otnw be aobrsebirgn htnynagi in the PCT ni soanfn'ic me.rdnoys ongoiLk at oyaekmi,aphl yn,arahemtopi adn htooiaphehpamysp wn.o maykleiHopa nt'ac eb cotrrec eaecusb neve ougthh tpsouiams is lots it iwll eb aerdebsbro ta teh atrle itkhc sagcdneni ploo dan fi tath onetds maek ,eesns the obyd illw uadjst rfo lwo murse simsouapt utb cngaaitivt the /K+H+ pmpu no l.csel tI n'tis nmeraahypoti uecbsea at hte iocgnllcte udtc pcinalpir l,csle porrebstaion lliw crco.u iTsh aevesl ihpatmophyoeashp sa the cocrert adn ylno nsewra cce.ohi

imgdoc  by* +2  
larascon  Excellent explanation, thank you ! +1  
waterloo  It's worth mentioning, that 25% of Na is reabs in the thicc ascending limb. 67% of filtered Na is reabsorbed in the PCT, whereas 85% of phosphate reabsorbed in the PCT. So pt more likely to show Hypophos. +  
coconut  UWorld QID:7626 says lab results in people with Fanconi syndrome will show hypokalemia +  
j44n  coconut i thought that too but then i remebred fanconi is associated with hypophosphatemic rickets/ VIT D resistant rickets and this little dude is going to be growing because he's 5 and thats gonna increase the demand for phosphate in his body +  


submitted by celeste(84),
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lieWh eht mefetili skir ni the gnreale aplnootipu si tusj woebl 1%, ti si %5.6 ni igrfetsrd-ee ersiavlet fo ntisaept dan it eissr to more ntha 04% ni ngzoycmooti intsw of etffcaed eep.olp angAlziny clsaics ditessu of eth sctngeei fo izensarophchi oend as yelar as in s93,10 iscFher udlscecno thta a oeornncccda aetr rfo siohcpsys fo boaut %50 in goioycnomzt tiwsn mssee ot be a ictalires mteist,ea hhwci is agiltnciisyfn grhehi naht ttah in ycidotizg wints of tbaou –11%90 (ee4Con.gl3Mmar2ni3b/9c5crPliv/m#6//tihn.6csp.f)

imnotarobotbut  How is one supposed to know this before having read this article? +33  
imgdoc  This question falls under the either you know it or you dont category. It isnt in FA or Uworld +  
jaxx  So why would these A-holes put it on there as if prepping for this exam isn't stressful enough :-| +7  
doodimoodi  Lol just why seriously +2  
champagnesupernova3  This was mentioned in the Kaplan behavioral videos +  
usmlecrasher  and there's so much unnecessarily BS instead of real questions +1  
j44n  I'm just glad we're seeing this garbage now instead of having an aneurysm in the prometric center +2  


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It dsai ti swa altfa ot aslem in ,etruo dna teh uiqesotn eskad aobut eilv norb fin.fsgpro eiSnc hte esmla r’aent bnegi onrb ni eth ftirs al,pec I sida %05 elesafm nda 0% a.msel

hungrybox  fuck i got baited +31  
jcrll  "live-born offspring" ← baited +23  
sympathetikey  Same :/ +  
arkmoses  smh +  
niboonsh  why is it 50% females tho? +2  
imgdoc  felt like an idiot after i figured out why i got this wrong. +2  
temmy  oh shit! +  
suckitnbme  This isn't exactly right as males can still be born as evidenced by individuals III 6,9,11. This basically an x-linked recessive disease. A carrier mother can still pass her normal X chromosome to a son (50% chance). It's just that the other 50% chance of passing an affected X chromosome results in death of the fetus in utero. Thus all males actually born will not be affected. +2  
makinallkindzofgainz  @suckitnbme, Correct, but if you're a live-born male, you 100% for sure do NOT have the disease, so the chance of a live-born male "being affected" is 0. +3  
spow  @suckitnbme it's not X-linked recessive, otherwise every single son would be affected and therefore have died in utero. It's X-linked dominant +3  
qball  Jail-baited +  
srmtn  correct @spow affected females= X linked Dominant +  


submitted by cantaloupe5(77),
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Tish eon aws ckytir btu I nhtik uoy vedlocu’ eond htis oen houiwtt eklodgenw of AMDN rt.creepso eSmt told ouy thta ltgaemuta ectasitav thbo Nno-MAnD dna MDAN trporsece tub it vcieattad onyl NM-noDnA spetercro in the eaylr he.pas htTa samne NDMA rocertpse ctiteava tearf MnDnoN-A osepecrtr. aTht manse msniohteg asw deiygaln AMND rpcrteeo ttvacangii nda hte ynol saenrw tath edam snese sa eth Mg hibiiginnt DANM at grsinte n.ioetptal eOnc the llec is lpziodredea yb oNMnnDA- tserepr,oc MNAD rcepotsre anc eb ectv.tdaai

hungrybox  I forgot/didn't know this factoid and narrowed it to the correct answer and a wrong answer. Guess which one I chose? +14  
yotsubato  >That means something was delaying NMDA receptor activating and the only answer that made sense as the Mg inhibiting NMDA at resting potential. What makes the fasting gating kinetics choice incorrect then? +5  
imgdoc  NMDA receptors are both voltage gated and ligand gated channels. Glutamate and aspartate are endogenous ligands for this receptor. Binding of one of the ligands is required to open the channel thus it exhibits characteristics of a ligand channel. If Em (membrane potential) is more negative than -70 mV, binding of the ligand does NOT open the channel (Mg2+ block on the NMDA receptor). IF Em is less negative than -70 mV binding of the ligand opens the channel (even though no Mg2+ block at this Em, channel will not open without ligand binding. Out of the answer choices only NMDA receptors blocked by Mg2+ makes sense. Hope this helps. +6  
divya  sweet explanation imgdoc +  
lovebug  really~~~ sweet. thankyou :) +  


submitted by imgdoc(135),
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I kniht sit the tghri LFM ra(ea C,) ton eht irtgh csduanbe uleusnc atth is dloiesen on het csros ocsein.t If teh sbnducea selncuu erew esoledni esh o'wnltud avhe citundabo in hre ftel eye. heT LFM idaeetsm csros ltka weebnte eth asncedub lcuuesn no boht ediss to the MFL no het eppsioot deis 2( nsudecab enicul, 2 MFL oen no ahec )ide.s In hre es,ac her hrgti FML sa'nwt ftgninoicun echne hyw ehs was anzigg left utb ehr tgihr ddilme serutc aw'nts cantcongti ot tadmiee rwtaeldf g.aze

iTsh itpcure p:eshl o.pahh4s/etut20w/p-g/ttw9bw:n-ochlFpm-cmo_.onet/2hm0nhdtpap//1w1utj/5

imgdoc  *medial rectus +  
quip13  This shows the MLF pathway in a saggital cut: https://ars.els-cdn.com/content/image/3-s2.0-B9781437719260100128-f12-08-9781437719260.jpg +