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Welcome to jotajota94’s page.
Contributor score: 13


Comments ...

 -8  (nbme24#47)
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A godo awy ot khnit uatbo htis si tath gionniaenst II is sprneet at eht NIIGA pes.ctoerr If yuo give an osnagentnii trcopree lckrobe hcus as taalr.osn tihs wlil lisaecdp the noegtnaiins II reetnsp in hte rrpotesec. uhts sicinnrgae eth lvseel of tsnoingenai II in umer.s


 +1  (nbme24#15)
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enenicdci = neburm of wen ceas/s wen a+sces tpaooulpni at rkis. new ssec=a .520 lPepoe at sirk htta aeyr 0055 (uinicgndl wen a.csse In oen nopti yeht were a anltpoouip ta rs)ik 52/0 055=0 %.49


 -6  (nbme22#48)
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I htikn teh key ot dusngranenidt tshi etsqnuoi is ahtt teh yob ash oeabv veearag ingLeept.h hit utsiealstm het laeerse fo HG (ng/1w./i0mwn2ludmphiws/:eoht2.bbcn.2v/5pt.81 hdie)rnlC with hgCsiun monesdry avhe htrso trseuat nbl/9hvgo56//h.3w7sdibn/.mt.tmiwu.epn:6pc13wnn) luiis hbnsiiit HG eeirotsnc c6.ew//M4npcvoraw/5.6.1.lmnsbp:tihnitlP/gC/h2m/tsiwcI ) nhtki htat bhto a ghhi icaorlc tide ni saft dna leouscg anc ekma uoy fat so uyo cna nelamitie etsoh neawsr hcicse.o


 +14  (nbme20#1)
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eUs hte neyger-iarbdHW ituaqeon

  1. Take eht qraues orot of 61,0/10 nda ahtt liwl eivg uyo eth nrqufeyec fo het vseesirce llalee = /104.
  2. laualectC het qcefnreyu of hte nimdoant lleale twhi =,qp1+ chhiw is p= ..9750
  3. Tyeh rea ltinleg uyo to ecaltulac eth reycqfeun of het sasidee crrer,ais hwich is thwi het qeniauot p2q.
  4. eyhT nwat nlyo teh sdeiase siarercr ni hchwi idoenlte si srtpene. oT aucteallc s,hit eus teh q aevlu 41()0/ nda mlpyiult by %08 ni itsh shdulo vgie ouy 200..
  5. Fanyil,l claacutel fro 2qP2 .(=02)5()790.0 0.04 = /521.
yex  Nice! ...and we are supposed to read the stem and do all this in a minute or so? :-/ +22
charcot_bouchard  Allele frequency 1/40. so carrier freq 1/20. 80% of 1/20 is 1/25 (80/100 x 1/20) +14
dickass  Ah feck, 2pq got me +
hello_planet  A handy shortcut for Hardy-Weinberg is that you generally can assume p ~= 1 if q if fairly low. It also tends to be easier to work in fractions if the answer choices are in fractions so you don't have to bounce back and forth between fractions and decimals. So with that, you send up with 2pq = 2 * 1 * 1/50 = 2/50 = 1/25. +4

 -8  (nbme20#10)
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neWh noeugacdjt lbiriibun BC() is less ,nad 3%0 of atlto -bril&niibg---u;t dasienecr hte cinourdpto of nnodcegujuat uibrnbiil or eddscraee paket.u ilmyeosHs is hte piobslse naresw ehcico.

BC si 3%05-0% rmlubriieapixnimeh-byi x.E( ptitiesaH ro h)coCAolBll is eomr ntah 0%5 -;g-&t Osoncibttru (itanr ro h)eraacipttex


 +5  (nbme21#5)
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DPA wlsfo rofm raoat ot puyaolnrm yatrer cinsedegra erlfeta.oeoeh rdTfra adcaric ututop sceianser

seagull  doesnt pre-load also decrease which would drop the C.O.? +
hungrybox  @seagull I think it would increase preload b/c more blood is going into the pulmonary arteries -> lungs -> pulmonary veins -> eventually more blood in left atrium/ventricle -> inc preload +50

 +3  (nbme21#42)
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iHgh lesougc dslae ot remo suinnli ioponuctrd ni eht ftseu lcle(ar htta the ernoohm inusinl is anlobc)ia -tg-;-& eglra tuefs l-o9&bg-;z(-,t)1 sebrmlop in olba.r

tinydoc  I thought that was technically a problem with the fetus and wasnt considered an obstetric complication. +3




Subcomments ...

submitted by radshopeful(17),
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hcncior ykinde aedsies --g&;t dreceeads EOP -t;g-& aceersdde cntichrotharimeco edknyi eidseas &t-g;- cseareedd 4PO- ioernetxc t;g-&- aniedscer ToiPh cHrnc ndiyek essiade -;t-&g ddeerecas 152, thryvdDiido icaicl(tlr)o t&-g-; rndaeesci TPH

jotajota94  she is also volume overloaded. more fluid leads to a decrease in Hematocrit. +5  
h0odtime  I thought PTH increases 1-alpha-hydroxylase which increases levels of 1 ,25? +1  
batmane  it's supposed to in the setting of proper kidney fxn +2  
h0odtime  Whoops +1  
avocadotoast  The decrease in GFR leads to decreased delivery of 25 dihydro vit D to the PCT and the decrease in functional renal mass limits production of 1a-hydroxylase. The increase in PO4 also stimulates FGF-23 from bone, which inhibits PO4 reabsorption and 1a-hydroxylase. +1  


submitted by beeip(123),
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hisT hsa nbee a utgoh ptcecon fro me to gt,e tub I ntikh m'I aiynlfl e:hetr

ehT semt si dnbicgeris pamryir naaelrd efiuicifcnnsy, or sdoAs'id.n

  • HATC si ngebi opdordrceuv-e to ieamsutlt hte sranedla ot ocedrpu cotolr,is but htye nac't rde,snop teerhi eud ot pyotrha or snrutdicoet (BT, neioatu:mum 4,DR ce.)t
  • eTh first 13 imoan iscad of THAC nca be eevdlac to mfor MS,-Hα icwhh malssetuit lsemnoateyc, cnauigs yihnimgtpreaentpo
jotajota94  Good job! Also, cortisol is involved in maintaining blood pressure. which was decreased in the patient. +7  
tinydoc  Decreased Na and increase K+ --- Hypoaldosteronisim Hypoglycemia, and hypotension --- Hypocortisolism so the adrenals arent working ---- adrenal Insufficiency the Hyperpigmentation comes from the increase ACTH as ACTH is from Proopiomelanocorticotropin. SO - increased ACTH also increases a -MSH ---> Hyper pigmentation. +10  
hungrybox  thank u for this answer +  
bilzcop  Ugh! I misread the question and chose ACTH :( +3  
cienfuegos  @bilzcop: same +  
cienfuegos  @bilzcop: let's never do it again, k? +1  
maxillarythirdmolar  Why does this patient have elevated BUN and creatinine?? +2  
lovebug  @ maxillaryhidmolar > I don't know exactly. but maybe.. Low hypo-adlo -> our body lose water -> hypo-volemia -> Decreased GFR -> Increased Cr,BUN. If I'm wrong. please correct me. +  


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I hknti it ahs tgmsinhoe ot od htiw gcinley d(eu ot ist mslla szei it nac fit ni nyma splcae rehwe horte oniam cidas acn tno and nhcee ti eiposvrd ulrrs“tcatu cpmanceosst” to teh lgen,alco ..ie upt a ikkn ni the ahalp hi)lex. If lcegiyn is lmscpadei yb tisnghemo eles, I dot’n htikn oarneplo-gcl acn mrof sit rtrecco socedanry curutert.s

jotajota94  True! also, glycine is 1/3 of collagen alfa chains, so it makes sense that substitution with alanine (which is much bigger) would lead to disruption in the alpha helix formation. +  
jotajota94  True! also, glycine is 1/3 of collagen alfa chains, so it makes sense that substitution with alanine (which is much bigger) would lead to disruption in the alpha helix formation. +  
thepacksurvives  Glycine is small and bendy, which allows it to form the fibrils for the triple helix +  
brasel  Also in general (FA 2018 pg 50) OI is from problems forming the triple helix which is secondary structure. Fortunately, they gave us something to reason with in the question (Gly->Ala) +2  


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I htink ti hsa hiongsetm ot do twhi gelniyc ud(e ot tis mslal zeis ti nac tif in yamn aplsce whree rheot aoinm dcais acn ont nad eehcn ti rpidsevo “tsatclruru sonmtpas”cce to eth locla,eng .i.e utp a ikkn in hte hpala )l.hiex If leigcny si cpadmlsei by hitmgoesn sel,e I do’nt htnik lcrneaog-lpo anc frmo its retrocc sdcoaenry rtesucur.t

jotajota94  True! also, glycine is 1/3 of collagen alfa chains, so it makes sense that substitution with alanine (which is much bigger) would lead to disruption in the alpha helix formation. +  
jotajota94  True! also, glycine is 1/3 of collagen alfa chains, so it makes sense that substitution with alanine (which is much bigger) would lead to disruption in the alpha helix formation. +  
thepacksurvives  Glycine is small and bendy, which allows it to form the fibrils for the triple helix +  
brasel  Also in general (FA 2018 pg 50) OI is from problems forming the triple helix which is secondary structure. Fortunately, they gave us something to reason with in the question (Gly->Ala) +2