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Welcome to potentialdoctor1’s page.
Contributor score: 34

Comments ...

 +10  (nbme24#14)
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plev-au rfeesr to the rbptbiiyloa of agimkn a ptey I rroer yirtpbabl(oi of anhivg a elasf itspove)i. nheW hte 5%9 cdoeciefnn vaeinlrt soed tno deiucnl the nllu eavul (1 for roa,it 0 ofr nceierd)ff,e 0 l;t& p ;lt& 500. ewbeent( %0 dna %5 acenhc fo viahng a laefs p)oesvt.ii w,vHoree henw hte 9%5 cnniecfdeo tavelnir cseuldni the nllu vel,ua 5.00 lt;& p ;l&t 01. ewteben( %5 dan 01%0 cecnah of ganivh a lsfea vitiosp.)e

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drippinranch  Excellent explanation! +2

 +7  (nbme23#38)
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rfnemIdo snecotn huldos eb ianbeodt by a piedrvor how hsa ieusfcfnti klndeeogw ot egiv na cutraace ospiretidcn fo eth ,entntinvroei het ksri dan t,bsinfee eanltatievr tatnesretm nda to easrnw lla fo eht epnsitt'a snuiosetq

stinkysulfaeggs  Hate this question though... the first thing you would have to do in this situation is refuse to do what the resident asks you to do. Then you could accompany them.... +10
peqmd  If not for the additional "refuse to sign consent". I think the answer would have been extremely straightforward. +1
dyckim4  I was taught that that the person who is operating should get the consent that's why I got this wrong.. +1

Subcomments ...

submitted by happysingh(45),
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o,s the Kye sdowr ttah on one si nmntiinoge : uoainmtgcmnic shoarclehdypu

the htpyshpoa eosg ekil hits :

na myatoiflnmar sitegnt .i.e(, canirubhsdao meogehhar)r idely srfsioib / garcrisn of eht ncriaaohd lngsaruainot >=; ripdmaie CSF nargeiad

teh eyk otnspi / npeccsot heyt era tinygr to ttes heer : .1 do yuo okwn thwa icoamuintnmgc puosrhahyedlc ih(owtut htme igelltn uyo oetsh wsrod) 2 . od oyu kown twsha' het altgopoyshiypoh o(f tioaungnmiccm hlchuraoyespd) si ?

potentialdoctor1  Exactly. To add to this, communicating hydrocephalus can be subdivided as follows: Normal-pressure hydrocephalus: Chronic/gradual decrease in CSF reabsorption at arachnoid granulations, usually due to calcification due to aging. CSF accumulates slowly, so ventricles are able to widen without causing an important increase in intracranial pressure. Symptoms occur due to compression of periventricular white matter tracts ---> Wacky, wobbly, wet High-pressure hydrocephalus: Acute decrease in CSF reabsorption at arachnoid granulations, usually due to inflammatory state in the subarachnoid space (eg, meningitis, sub-arachnoid hemorrhage). CSF accumulates suddenly, causing an acute-onset increase in intracranial pressure +7  
sunshinesweetheart  not to take away from your perfect explanations, but if it were a woman with neck stiffness and fever (rather than circle of willis rupture) that could lead to increased CSF production, right? I think that's the only case where CSF production would increase. Also I think decr absorption in arachnoid granulations in that situation as well so it'd be a shit question +  
peqmd  If anyone like me also got "decreased absorption in choroid plexus", as their wrong answer it's because the choroid plexus doesn't "absorb" it produces. +9  
alienfever  FA 19 p510 +2  
alienfever  If anyone chose F, communication hydrocephalus is caused by decreased absorption and not increased production. FA 19 p510. +1  
an_improved_me  So she has a leaking aneurysm for how long.. gets it repaired, and then within 2 days has an inflammatory response that leads to decreased CSF absorption at arachnoid granulation... Is it the bleeding associated with the aneurysm causing it? The surgery? I'm inclined to say the latter, given that it happens coincidently after the surgery, and not for however long it was leaking beforehand. Thats what tripping me up. +  

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potentialdoctor1  Might be relevant to add that there are two beta-blockers that are actually partial beta-agonists (exert their blocking effects due to lesser effect when compared with endogenous catecholamines) ----> Pindolol and acebutolol +10  
thelupuswolf  Key to note as madojo said that a partial agonist will compete with the full agonist in the presence of the full agonist, preventing the full agonist's maximum effect +