Welcome to step420’s page.
Contributor score: 33
I don't think HR would explain the grade 2/6 murmur, but SV would
is the AIDS to indicate that he can't get live vaccinations?
Yep, also his unimmunized cousin points you to the fact that he got it from someone else who was unimmunized.
ugh, I saw Ca2+ and stopped thinking.
MAGIC RACKS is a good mnemonic ive heard for 450 inhibitors
(macrolides, amiodarone, grapefruit, cimetidine, RITONAVIR, alcohol (chronic), cipro, ketoconazole, sulfa
Macrolides EXCEPT azithromycin -- they like to trick you with that one.
chronic alcohol induces 450, acute alcohol inhibits 450
O-linked glycosylation of secreted and membrane bound proteins is a post-translational event that takes place in the cis-Golgi compartment after N-glycosylation and folding of the protein
I was wondering the same thing because doesn't androgen insensitivity also have normal female secondary characteristics. Was it the levels of hormones because she doesn't have abnormally high testosterone?
Androgen insensitivity has the same presentation and symptoms. What's the clue that it is mullerian agenesis instead ?
Testosterone would be high if it was androgen insensitivity
FA 2019 Pg 625
Testo would be high in AIS. in AIS pubic hair, axillary hair doesnt devlop because of androgen insensitivity. both have normal breast dev and primary amenorrhea
This is not androgen insensitivity because she has perfectly normal Estradiol, which means she has perfectly normal ovaries. She also has regular female levels of testosterone.
Above answer is incorrect because hyperplasia can be either physiological or pathological. Prolonged hyperplasia can set the seed for cancerous growth however.
Stated another way, in pure hypertrophy there are no new cells, just bigger cells containing increased amounts of structural proteins and organelles. Hyperplasia is an adaptive response in cells capable of replication, whereas hypertrophy occurs when cells have a limited capacity to divide. Hypertrophy and hyperplasia also can occur together, and obviously both result in an enlarged (hypertrophic) organ.
FTR Pathoma Ch 1 Dr. Sattar mentions hyperplasia is generally the pathway to cancer, with some exceptions like the prostate and BPH.
Tubular hypertrophy is the natural compensation post renal transplant. Just one of those things you have to know, unfortunately.
Isnt Kidney a labile a tissue & thus should undergo both. This ques is dipshit
Dr Sattar says, kidney is a stable tissue, at least pct is as seen in ATN.
But I read, basically kidneys are mostly formed whatever number of nephrons have to be formed by birth, after that they can only undergo hyperplasia aka increase in size/or regenerate if need be in case of atn. We cant have more number of nephrons.
@brbwhat , do you mean kidneys can only undergo hyperTROPHY?
bHCG directly increases testicular aromatase activity, it's not because of the increased amount of testosterone.
And for those who were wondering (cause I was), Sertoli cells have aromatase (FA 2019 p. 614)