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submitted by keycompany(310),
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oypreviHttenlani dereesca 2CaOP. lernaCt moeotspehcerrc onperds to olw OP2Ca by tsrgosicvaotinnc rcreaebl odblo slsseve.

)A Araretli Blood gnOexy tatienncnoCor: odBol gOynxe Cnneocotiatnr si tceldiry lteaedr ot bH eocnnctnrotia and sirauntota ()2OSa ,A19F02 p. 356. Via het oBhr efEctf, ceseddrae a2CPO llwi neiarsce ,SO2a tsuh inagsnrice oblod eongxy ecnntotnro.cia

B) ateliArr ooBdl PO2: P2aO ashngce in nposerse ot ddescreea PO,A2 ,PIO2 ro isud.iffno eTreh wolud be no gehanc ni Pa2O ngdiur ittylpaionnveerh crihoal)(e.ttely

C) eatriAl rursseeP: seaDreecd 2OCPa is ocsataesid iwth nict,oosvcriostan wihhc wluod sieceran bdolo .euprress

)E lCebarer uTiess pH wuold recesain ued to sierrpytora .laisalsko

keycompany  EDIT: Via the **Haldane Effect**, not the Bohr Effect. +  
impostersyndromel1000  excellent response +  
teepot123  fa 19 pg 489 +  


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elPsae cna ouy nkydil esahr NMBE 22 nda 32 Qissuonet htiw em . I lnoy see het rawsesn tbu I od nto evah the ulfl okiau?Thnenss tq in ncvaeda

makinallkindzofgainz  I think you may have this website confused with nbmequestions.com +7  
teepot123  why don't u buy the exams like the rest of us? +2  


submitted by chosened(2),
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Ccorrte me if I m ognrw utb I inthk sti' elyrglana i.pootmslplsaia Plsplamaio can epdovel awenyreh alnog eht tpisoyrrrea ,arctt utb sotm fnteo ffctea het rylaxn nda eht olvca orcds narg(eyall ta)aslp.po ioimsNtlo ruse ohw YH htsi si but shere Moer :niof nmps-aoesaierr/ueea//pr-ayeop-sso/r/s:pdaise.rehleaciisrtestrotrdlrrtgssriat

hyperfukus  yes you are definitely correct i think its a common wtf q that pops up bc there's one on uworld that asks if its true or false vocal cords and i had to hunt my prof down to figure it out... +2  
hyperfukus  also i think they love anything that compromises the airway +  
winelover777  Shout out to Pathoma, Respiratory chapter, Larynx section, Laryngeal Papilloma heading. Only reason I got this question right. +1  
teepot123  yep dr sattar nails it ^ +  


submitted by seagull(1571),
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If uoy o'dnt onwk thaw iaomclruD soed lkie ayn aonlrm .hmuna heT uscof no hawt risnaip toends' ,od lymnea 'ist noedst' cftfea PT tmie nad msto ilpsl d'not sceaerni ocitltgn pl(eiycseal hwit )rip.nias sihT is woh I logci ot hte tirgh nrew.as

usmleuser007  If that's then thinking, then how would you differentiate between PT & PTT? +25  
ls3076  Why isn't "Decreased platelet count" correct? Aspirin does not decrease the platelet count, only inactivates platelets. +4  
drmohandes  Because dicumarol does not decrease platelet count either. +  
krewfoo99  @usmleuser007 Because the answer choice says decrease in PTT. If you take a heparin like drug then the PTT will increase. Drugs wont increase PTT (that would be procoagulant) +3  
pg32  I think usmleuser007 and is3076 were working form the perspective of not knowing what dicumerol was. If you were unsure what dicumarol was, there really wasn't a way to get this correct, contrary to @seagull's comment. You can't really rule out any of these as possible options because aspirin doesn't do any of them. +4  
snripper  yeah, it wouldn't work. We'll need to know with Dicumarol is. +4  
jackie_chan  Not true, the logic works. You gotta know what aspirin does at least, it interferes with COX1 irreversibly and inhibits platelet aggregation (kinda like an induced Glanzzman), all it does. PT, aPTT are functions of the coagulation cascade and the test itself is not an assessment of platelet function. Bleeding time/clotting time is an assessment of platelet function. A- decreased plasma fibrinogen concentration- not impacted B- decreased aPTT/partial- DECREASED, indicates you are hypercoaguable, not the case C- decreased platelet count- aspirin does not destroy platelets D- normal clotting time- no we established aspirin impacts clotting/bleeding time by preventing aggregation E- prolonged PT- answer, aspirin does not impact the coagulation factor cascades in the test +3  
teepot123  di'coumarin'ol +  


submitted by armymed88(47),
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oGcsuel is ronaotd-ptcesr oint eeottycsren fo SI vai suiomd

toxoplasmabartonella  That makes that glucose needs to be given with sodium. But, what about bicarb? Isn't the patient losing lots of bicarb from diarrhea? +3  
pg32  Had the same debate. I knew glucose/sodium was the textbook answer for rehydration but also was wondering if we just ignore the bicarb loss in diarrhea...? +4  
makinallkindzofgainz  @pg32 - Sure, they are losing bicarb in the diarrhea, and yes this can effect pH, but it doesn't matter that much. You're not going to replace the bicarb for simple diarrhea in a stable, but hydrated previously healthy 12 year old. You're gonna give him some oral rehydration with a glucose/sodium-containing beverage. Don't overthink the question :) +2  
makinallkindzofgainz  *dehydrated +  
teepot123  salt and sugar, that's all the kid needs when ill simple +1  
mtkilimanjaro  Hm I put bicarb/K+ since thats lost in diarrhea, but I think the key thing in this Q is that its only 6 hours of acute diarrhea and nothing else. You would prob give bicarb and K+ in more "chronic" diarrhea over a few days or longer not just a few hours +1  


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The imciodstlur penrigrsfne plaha toxni is a eniasclhtei ihwch alcseev thlencii ot cplnohroploheyhsi dan gce.yiledrid iats,eEyllsn hpala tixno sicmim ppsshihpooela C. Tsih snmea it sah a euaglvy ilsrami eftfec fo eht ppehhsopissola nees in aslciluB sueCer and aLrieits Meceogononsty. ehT edn ulerst of het notix ciiattoavn si ioitcanavt of odsnce seenremsg yssmets ugorhht eygcelrdidi KAA( i)rlocyald,ecgyl ihhcw aastvteci srleaev sapyah,tw omst otanylb in hsit csea dAihocranic acid easoimbtlm nad 8L-I, wthi the tne cfeeft of aeeridsnc vsrcaalu tiereilabmyp gaenlid to e.aemd

teepot123  fa 19 pg 138 +1  
faus305  alpha-toxin is also known as lecithinase. I got this right because I remembered the sketchy. That being said, can someone explain how this possibly received a "13.1 difficulty score?" It does not seem like that easy of a question. +  


submitted by tissue creep(113),
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Lfingti deha liehw onrep: 1 ooacthilS nm mesli: 2 onmsCg:othoni 2 shomnt

pg32  Where do you guys learn that cooing starts at 2 months? It isn't in first aid or boards and beyond so this was an annoying question for me +1  
drschmoctor  @pg32 From being a parent! Otherwise little chance I'd remember all these milestones. +4  
drzed  I'll get right on that @drshmoctor :). If only I could have a kid to memorize all these damn developmental milestones. That would make life easier haha. +6  
snripper  Yeah, I don't see cooing anywhere. +  
teepot123  thankfully a lot of my friends on insta keep posting pics/vids of their babies reaching milestones so im well updated lol +1  
pjpeleven  Mnemonic: "Coo at Two" +1  


submitted by trump2020(6),

You guys are way overthinking this. Chronic hypoxia causes hypertension due to SNS activation and EPO production.

teepot123  yep +  


submitted by hayayah(1079),
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lAghothu alfh ethes meshoorn anc lucaytal slao eb crsdeete ormf teh umoeddu,n hte mouddenu si satadesoic eth omst hwti CCK eslr.eea

didelphus  Gastrin, intrinsic factor, and pepsin are secreted by the stomach. VIP is synthesized in neurons, so CCK (from I cells of duodenum) would be most directly affected by a duodenectomy. +22  
teepot123  fa 19 pg 365 +  


submitted by pparalpha(84),
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hinbitnoii of tuliocrmebu raozpoltineimy → iinihbtsi tsgcyaospiho of truae atls,crsy oeeclkuty civaiatnot nda riomi,tnga adn clel msa.ehxocti

teepot123  fa 19 pg 476 +  


submitted by ihavenolife(69),
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dkI if it peshl btu I tgo it siugn het toH oeBn-T EKAst mmicenno

Hto -&tg; LI 1 (ev)rFe

T elcl tmoluanstii &-tg; LI 2

Bneo rawmor snlitamoiut ;-tg& IL 3

gIE &g;-t LI 4

gIA ;g&t- LI 5

aKetu epsah nreotip t-&g; LI 6

temmy  cyclosporine inhibits transcription of IL2 +14  
mysticsoul  FA2018 Pg 120 +3  
teepot123  same page also for 2019 edition^ +  


submitted by haliburton(214),
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FA 2071: crSetee tnctafrasu rmfo rlelaalm id.ebos oAsl evser as cruosserrp to yept I eclsl nda ehrot pety II l.elcs tfoeaelrPri rigund nlgu m.eagad

teepot123  fa 19 pg 647 +  
moms  thanks! +  


submitted by tinydoc(233),
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troeachuNpi nPai rtafe oktrse is actnlre stoP eksort ianp orenySdm

deusac yb etoltarnlcraa ltamiach olsisne

.gP 405 9AF1

chandlerbas  agreed! more specifically damage to the VPL +6  
docshrek  Pg. 403 FA 19. +  
baja_blast  Both commenters above got the page wrong; it's FA 2019 p. 503. +4  
teepot123  looooool ^ what were the odd of both being wrong +  
mumenrider4ever  Pg. 515 FA 2020 +  
bbr  503 in 2019 Interesting that its seen in 10% of strokes. Starts with allodynia ---> neuropathic pain. +  


submitted by hungrybox(1040),
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tlpysDsaic nive era a orrupcers ot m.laaonme heyT ehav a,rlueigrr scal"istdy"p eb.sdror mrbeReme het ""B in ADCB sdtasn fro rulgirrae srodB.er usveN asmne .lmeo

hretO w:ssrnae

  • shcsnaiota srganinic - ginraeDkn fo isnk scsitadoea hwti yTpe II btdsaiee esitllum

  • blaas lelc cmaorncai of nkis - el,rayR fi ever masetais.zset ooymCnml afescft upepr ipl.

  • eulb senuv - lrlcBeedou-o etpy of momnoc l.oem nni.gBe

  • gimneedtp hocrsirebe rtssaekoi - tck"Su "on a.aeapprcne lMysot being.n effstAc eodlr epe.lpo

  • Nt(eo - you ulsluya ees yonl no.e fI letumilp berhercois steaokrse rea n,see it aiscenitd a GI nylagnamic - aak eLtrs"ée-Tral is)ng
usmleuser007  correction ~ BCC affects the lower lip more than the upper +1  
sympathetikey  Pathoma says upper lip, good sir +26  
hungrybox  Yeah basal cell carcinoma actually affects the upper lip. Counterintuitive because it's "basal" which seems to go along with the lower lip. Here's another source (this website is fucking gold btw): https://step1.medbullets.com/oncology/121593/basal-cell-carcinoma-of-the-skin +5  
pg32  Can anyone explain how we can rule out C or E purely based on the question stem? If we read into the question that we are looking for something related to melanoma, then I get why we can rule out C and E. However, the question simply asks which lesion appears on both sun-exposed and nonsun-exposed areas of the patient's skin. I would say that C, D and E can all occur in that distribution pattern. +5  
paperbackwriter  @pg32 because it specifies "this patient's skin," and the only ones he is more likely to get than the average person because of his family history are dysplastic nevi +2  
teepot123  fa 19 pg 473 +  
rockodude  just remember BS. basal cell upper, squamous cell lower +  


submitted by usmleuser007(397),
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SNC g/ehHrraoem a1m )edga 214-8 rHs = rde oneu s)r2n 13- sdya = ulieporhstn filiecuevq(at ro)eis3s )cn -35 = cegahmpsrao r4aogc) i()lmi 21- sekwe = ceaiRevt sloiisg (+ vlaasrcu ifo5)lrrentioap) eorm tahn 2 sewke = ailGl scra

teo(:n hte opgnasiehest is riilsma ot MI adn its rcas nrtmfoo;ai ,oevewhr hte etim rcuseo rfo CNS is ujts et)rfas

teepot123  fa 19 pg 500 +  
icedcoffeeislyfe  FA2020 pg 512 +  


submitted by hungrybox(1040),
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sreockriaoWK-enkff smyonedr due to hteiianm B1)( dy.iicecnfe momCno ni alhcsi.lcoo

eTh raneso hyw heyt isda leus"trs fo hoclaol adn udgr rencse rea gve"itena si ttha teh eeinitrdffal esindclu aetcu lloohca to.caiotinxni

'rieksenWc :drita

  • usonoifcn
  • aprslysia fo eye meslsuc ela*o(pmapohtg)hli
  • xaaati

ser*stpen heer sa antsymusg

orssKoffka' op:schsysi

  • yomrme lsos (gtaedaenror nad oadreg)etrr
  • ngkiam htis up cbu()nfiltonoaa
  • tnespyialro neachg
teepot123  fa 19 pg 559 +1  


submitted by mcl(599),
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Vibiro lroaeech si a er,eaivngm-agt cmmoa hepdsa atrabcie ttah can cueas rwyate rdrei.aha alheCor txnio isotcfunn by catitngvia eth Gs sieontpr t;-&-g secinrgnia ytctiaiv of anedlyly eyclacs -;gt&- neaicdrse PAcM --;t&g rsidcneae Na+ and C-l exuflf t--;&g eiad.ahrr

teepot123  fa 19 pg 146 +  
lovebug  Cholerae's exotoxin does not invade mucosa. but just permanently activates Gs according to FA. +  
lovebug  Same mechanism[Increase cAMP] : 1) Labile toxin of ETEC, 2) Edema factor of B. anthracis, 3) Pertussis toxin of B. pertusssis +1  


submitted by nerdy nik(10),
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TVD tath tnwe ot the i.barn fI ti luwod aveh ogen ot teh pmuonlary rayter hse ulodw veah a EP, tub it cessodr het atari allw aiv a eptnta nfoaerm eloav nad netw ltdcyeir ot hte bia.rn

cienfuegos  Some more UW info: incomplete fusion in up to 25% of adults: remain functionally closed until RA > LA pressure (e.g. valsalva), esp. concerning if hypercoagulable (e.g. OCP) -evaluate cryptogenic stroke with buble study: inj agitated nl saline and look for bubbles in left heart +2  
athenathefirst  Are you very sure of this answer? because she is an adult and not a baby anymore so it can't be a patent foramen ovale. +  
teepot123  has not closed basically +  
kevin  cienfuegos is spot on with his explanation @athenathefirst +  


submitted by hayayah(1079),
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hte yarjtoim fo ocranb eoiddix coelemslu are eradcir as patr fo the iboatceanrb ebruff .ysmest nI sthi ,ssmyte caobrn dxoeiid fidssfue otni eth .RCsB Crbnicao dyrasaneh )(CA hnitiw RBCs uqiclky vrotcens het rcbnao ixoiedd itno cicobnra icad OH.)C23( raCniboc dcai is an alestnub eteadintriem umeceoll atht mltmiieayde osadsiistec niot iabaeconbrt ison O3()-HC dan drnyeohg H()+ nis.o

heT nlyew siydzhtnese noraabecitb oin si senroattpdr tou fo het BCR tnio the alapsm ni aehncxge orf a doichelr ion ;)C−l( isth si aecldl hte lidehroc itsh.f enhW eth bodlo sechear het n,gslu het inacabbeotr ion si rtodarnpets kcab into het BCR ni cehagnxe fro eht erchldoi noi. hTe H+ oni soeicdsitas rmof eht eiomhobngl dna nsbdi ot the trneiboabca .oni iTsh opsdrceu hte ibcnocar dcai eeadrti,eimnt cihhw is dovcenret abck ntio robanc ixoided tuhrhog the tmiacyzne oitcan of CA. ehT brocan ddiieox eorddpcu si lleexpde hurhtgo the lnugs inrdgu in.leohaaxt

hungrybox  Amazing explanation. Thank you!! +1  
namira  in case anyone wants to visualize things... https://o.quizlet.com/V6hf-2fgWeaWYu1u23fryQ.png +5  
ergogenic22  CO2 is carried in the blood is bound to hemoglobin, known as carbaminohemoglobin (HbCO2) (5%), dissolved CO2 (5%), bicarb is 90% +3  
pg32  Nice explanation, but can anyone clarify how we know from the question that we are measuring HCO3 rather than dissolved CO2? +3  
qball  @pg32 This question is asking about what accounts for the LARGER amount of co2 and the HCO3 buffer is about 85% of this transport and dissolved C02 is about 5-7%. https://courses.lumenlearning.com/wm-biology2/chapter/transport-of-carbon-dioxide-in-the-blood/ +3  
teepot123  fa 19 pg 656 +1  
surfergirl  "majority of blood CO2 is carried as HCO3- in the plasma." I guess that is all they're testing us on, just in a very convoluted way. +  


submitted by beeip(124),
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I itmhg be het olny nepsro on arthe hwo tog hsit neo wo,ngr utb ss:eregaldr

T"IT slynaisa diulcnse veeyr ctuesjb owh si izeamrnddo acnicrdgo to dizremndoa tttrmeaen snnmgeista. It oingsre aionncel,mponc plortoco vtdi,naioes ai,alrhtdww dna ntynhiga hatt ppenhsa aftre nra.m"oztdiioan[1]

yo  You're not. I also goofed. +20  
seagull  https://www.youtube.com/watch?v=Kps3VzbykFQ This video is a pretty decent explination worth your time on the subject. +2  
hungrybox  I got it right but I was only like 50% sure. So I appreciate it. +  
drdoom  ^ linkifying @seagull: https://www.youtube.com/watch?v=Kps3VzbykFQ +2  
teepot123  ^ same video above used when I analysed my form 20 q which I got incorrect at time, its very clear at explaining this, helping me get it correct on this form +  


submitted by mcl(599),
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ntaetPi mya ahev dyrirehtae gd,ioenaame hwcih is oataecdssi whit r"nceerutr saakctt of teninse, esmsi,va ilodlacez usubcueaston admee ogiilvnvn het smxier,ietet ,tganiilea fec,a ro tnur,k or slcuombusa ameed fo eprpu yaiwar or slbow."e ehT eracitl gseo no ot sya 1eesea-rt"sC hnitibroi korsw yteridcl no the ptolenmcme dna cacnott mpasla asecsdac to dceuer knbianiryd raeeesl" hhwic si olsa apyrbbol doog ot .nokw

p..r6ptwcilohw/nMi/3n6:ngtC.mc/a.tls/6csem1w/i8/hvPb3

notadoctor  Thought this was a trick question as C1 esterase deficiency also results in a decrease in C4. However, the second answer choice was not referring to C4 but to C4 binding protein, which I now know is different. I also didn't realize C1 esterase was technically a complement protein. +4  
youssefa  Based on many sources hereditary angioedema does NOT cause a rash (urticaria) which is a main differentiating point between angioedema and allergy. This mislead me in this question. Any clarification? +22  
ergogenic22  +1 on the above because uptodate states that c1 esterase inhibitor deficiency, both acquired and nonhereditary, are both non-urticarial, non-pruritic, and that is confirmed by the above linked article +2  
sahusema  Question writer probably didn't know the difference between cutaneous urticaria and subcutaneous edema. +3  
almondbreeze  same. got it wrong bc the pt didn't have sx of hereditary angioedema - swollen lips and eyelids +2  
teepot123  fa 19 pg 107 +  
beloved_bet  According to Amboss "Mast cell-mediated angioedema Often associated with urticaria and pruritus Other associated with clinical findings of allergic reactions (see type 1 hypersensitivity reaction) Presents within 30 minutes to 2 hours after exposure and resolves over hours to days" +1  


submitted by soph(61),
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lla eth rheto tnoiosp pt uowld hvae ,tsrueu siear.ov urrtnes teyh otnuwld loko olamrn nad ehty uwdlo ahve ohrpcait seriao.v

turtlepenlight  This makes sense, but I was thrown off by the "normal-appearing" b/c wouldn't AIS pts not have pubic hair? +  
drzed  They just say it's a normal appearing 17 year old girl; not that the external genitalia are normal appearing. +1  
teepot123  fa 19 pg 625 +  


submitted by neonem(571),
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RPR = cnngreise test ofr lssiypi.h ihsT si a itveos-cglgkno pttenar ,rhsa acricsihtrtace of eracosydn pyii.slsh Drug fo ehoicc fro hlypsisi at nay egsta is umuastraclirn Pcliel.nini

ragacha  PAG 147 FA 2018 +  
teepot123  also says which antibiotic, only 2 in the list, helps narrow down, pen more commonly used +2  


submitted by hungrybox(1040),
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ewhn ffid iglsen rndats rpeira cmeimnssah are u:sde

  • rirepa nlywe nedehsstyzi tn:dsra tmcsiham iaerrp (Lchyn ers)ynomd
  • ariper niyierpimd ismerd uasdce yb tad VU :oxpueers ecolniudet oicensix eprari meoa(rredX umig)eonpmst
  • arpire ocnsueao/oxipntts rta:enoilat aseb eincsoxi eriarp
meningitis  Brca: recombinant repair +  
brotherimodu  P.40 FA2019 lists the different DNA repair mechanisms +2  
teepot123  fa '19 pg 382/3 +  


submitted by usmle11a(76),
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NIF y llwi ibhitni tennedffariitoi of hT2 slecl adn hrtoreeef e(a, )gowrb)n eoaurtgyrl T llcs)de fi eth scae wsa tkiglna boatu rusvi

teepot123  fa 19 pg 108 +  


submitted by aesalmon(83),
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Cna aoynen peinlax why iths is ton a rpiatityu ?admaeno sI ti stju a lakc of hetro yo?smptsm

benzjonez  I think that they just wanted you to notice the **calcifications**. Per FA, "calcifications are common in craniopharyngiomas," whereas I don't think you'd expect to see calcifications in a prolactinoma. +21  
epr94  also the option is prolactinoma not broad pituitary calcifications and he doesnt show any specific signs of high prolactin +  
madojo  It says hes coming in for constituional delay in growth and puberty, i took this to mean that he had low LH, FSH due to decreased GnRH from the prolactinoma. Why did they have to say low-normal serum gonadotropin, why not just say normal? fck u nbme +1  
madojo  It says hes coming in for constituional delay in growth and puberty, i took this to mean that he had low LH, FSH due to decreased GnRH from the prolactinoma. Why did they have to say low-normal serum gonadotropin, why not just say normal? fck u nbme +1  
ac3  @madojo I believe since suprasellar tumors can cause a mass effect on pituitary gland to decrease gonadotropin levels. Where as prolactinoma causes a rise in prolactin which downregulates gonadotropin secretion. Please correct me if I'm wrong +1  
teepot123  fa '19 pg 516 +  


submitted by egghead(1),
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shTi si oen fo othse ntsoeusiq I wsa evren gogin ot g.te tsI' not in A,F I to'nd ihknt ve'I eesn ti in ac.ssl

hungrybox  same :( +  
masonkingcobra  My issue was the stem said no skin damage (I would think pulling out your hair damages your scalp) [Turns out it does not](http://onlinelibrary.wiley.com/doi/full/10.1111/j.1529-8019.2008.00165.x) +  
gh889  FA 2019, pg 551 +8  
meningitis  Compulsively pulling out one’s own hair. Causes significant distress and persists despite attempts to stop. Presents with areas of thinning hair or baldness on any area of the body, most commonly the scalp. Incidence highest in childhood but spans all ages. Treatment: psychotherapy is first line; medications (eg, clomipramine) may be considered. +13  
step1soon  FA 2019 pg 551 +1  
teepot123  damn its in FA and Ive never ocne read it XO +  


submitted by strugglebus(165),
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rcnypolsieoC si a neirluaincc hirib,into hwhci msia ot rscedeea L-I2

teepot123  fa 2019 pg 120 +3  


submitted by killme(13),
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Ineinntot to Ttear lsisnyAa

usmleuser007  in a per-protocol analysis,[6] only patients who complete the entire clinical trial according to the protocol are counted towards the final results +1  
sympathetikey  "In an ITT population, none of the patients are excluded and the patients are analyzed according to the randomization scheme." +7  
smc213  This video helps https://www.youtube.com/watch?v=Kps3VzbykFQ +13  
rio19111  Thx smc213, really helped. +1  
trainingrats  Where is this in FA2019? +  
teepot123  the video explains it well, no need for fa +  


submitted by chillqd(39),
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itentsaP hwti umefagin /22 to Cdandai salcabni duohsl iecveer dan oEisdcnhiacnn talgnifaun nteag disa itccaepr egiesidunl

nEishdcainonc ihintbi hte ssheitysn fo taeb ),,Dc(3-1l-guan na einsaselt pmetnocno fo hte llec llwa fo pstuibecsel esupgirlslA scsipee dna andCaid cspiees

fanCiugnspo caPekga ntersI

teepot123  fa 2019 pg 200 +1