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Welcome to wutuwantbruv’s page.
Contributor score: 26


Comments ...

 +2  (nbme23#36)
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,X64X DDS deu ot esxvcesie nad iappranrtopei opusxeer to onecdrnagi doisesrt uigndr rylea gintestoa .i.(e aoginentcl ldnarea )yrsa.pphlaie

jboud86  I'm just adding the page number from FA 2019 to this comment. FA 2019 page 625. +4
leaf_house  FA 2020 p 639 +

 +3  (nbme23#31)
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hrtSitag mrof AF 0281 pg :630 Ctfle ipl dna fletc paltea vhea ,tstiindc luotifmliartac eiolgoteis, btu fonte crcou "te.regoth


 +5  (nbme23#1)
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hTe naehnbecoomhlrp uwodl be on eht taucal tuesf os tusj teg idr fo D.() Teh bkalc" heol" ttah eth fsetu si naliofgt ni si eht tnlsgatieoa cas os teg ird of (C). Nwo I am on tsolrunuad xerpte btu I nokw hatt teh mniatcoi atycvi auletelvny pedxans ot sfeu iwth the rcohion ryehbte netiilmngai the riihcoonc vitcay (.)B In emtsr fo erwhe eth tocniami ycavti si whnos in shit igae,m I ma tno ,uers os abyem mesoneo acn pelh tub sthi seleva eth kloy csa chwih lltapiycy aappser iintwh het ttaialeosgn cas uondar .55 ewe.ks

kateinwonderland  At the end of the fourth week, the yolk sac presents the appearance of a small pear-shaped opening (traditionally called the umbilical vesicle), into the digestive tube by a long narrow tube, the vitelline duct. (Wiki) +4
tallerthanmymom  But why does it look completely detached from the fetus? I eliminated yolk sac first because of this +
makinallkindzofgainz  If you look reeeeeeeally closely, you'll see some signal between the yolk sac and the baby. Although you can't see the entire connection, they are connected. +4
thotcandy  Pt is roughly 8 weeks pregnant so and typically by 9th week, Amniotic cavity has expended to fill entire volume of Gestational sac. So the entire black part around the fetus is GS/AC. +2
euchromatin69  and also by 7 week vitelline duct obliterates between umbillicus and yolk sac +

 +2  (nbme22#12)
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tCn'a be raohsαx1ey-ly7d seabuce siht uwodl enptrse thiw ieyhnnsterop nda omse rsot of uoismugab uaxsel ronepiattnse a(msl)e ro kcla fo noerysdac xelsau doneetmplev semf).ae(l n'atC be lyoyh1eβar1s-dx abueces siht udlow eensrtp twih the optpsioe fo the kid's tnirnesaotpe edu ot eth pudrconoti of ri-eorntceootx1cy1odes ismli(ar ctseffe to nesoalreodt btu ont yalern as eno)t.tp heT rehto two oulwd not elylar kmae eesns sncei teher are denesaric leeslv of nee1syxyedoohpgor7tr-r





Subcomments ...

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Im’ oiggn ot ioptn uto ttah a olrnam ehlhyta idk wiht no caaidcr rshoiyt or mmostspy nda on lfimya tyshori of dsnude drciaca htdae for a ptesorp-sr aylspcih is plabobry inogg to heav a ninbge xeam on etmtra wtah uyo nhtki yuo .hare

the260guy  What a weird question. I could definitely hear a fixed split heart sound. And it was loudest over the pulmonic valve too which makes it even more of a dirty question. But I guess what I was actually hearing was an S3 heart sound. +9  
wutuwantbruv  @the260guy I believe the splitting is being heard only during inspiration, making this normal physiologic splitting. Perhaps that's just my ears. +11  
angelaq11  don't have adobe and couldn't download it, so I just chose whatever, but your explanation suddenly makes me feel dumb but grateful! Loving your tips! @benwhite_dotcom +1  
blah  @the260guy Have to agree with wutuwantbruv. I interpreted this as a physiological splitting, had the opportunity to hear it in a newborn as well. +2  
prolific_pygophilic  Definitely S3. FA 2020 pg 287 "but can be normal in children, young adults, athletes, and pregnancy" +1  
dhkahat  I swear to god it wasnt just during inspiration but what the hell do i know +  


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Hwo si stih hte ewnars fi erteh si no ilfamy srotyih fo tnrecerru ascurfe?tr I gthhtuo oesnosiegets emeaipftrc was ouatoalms i?natdonm

seagull  Exactly!! it's an autosomal dominate disease! +10  
emcee  Autosomal dominant diseases are variably expressive. Still, I think this was a badly written question (should have given us some family history). +  
wutuwantbruv  Also, FA says that fractures may occur during the birthing process, which is what I believe they were going for. I don't believe these findings would be seen at birth with any of the other choices. +  
d_holles  Yeah I thought I outsmarted NBME by selecting Rickets bc it said no family history ... guess I got played lol. +9  
jean_young2019  Could it be a sporadic cases? Spontaneous Mutation This is a change in a gene that occurs without an obvious cause, in a family where there is no history of the particular gene mutation. OI is inherited as an autosomal dominant trait. Approximately 35% of cases have no family history and are called "sporadic" cases. In sporadic cases, OI is believed to result from a spontaneous new mutation. http://www.oif.org/site/PageServer?pagename=Glossary +6  
avocadotoast  Amboss says the severe subtypes (types II, III) of OI are usually due to a new (sporadic) mutation in COL1A1 or COL1A2, while patients with the mild forms (types I, IV) typically have a parent with the condition. +  


submitted by mousie(209),
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naC nesemoo aspele paelxni shit to ?me I 'dnto drunndseta wyh gnsttira teh ohtre gdur ldwuo ont uocnt sa cixneusol ircert?ia

seagull  This has to do with Intention-to-treat analysis. Essentially, when participants are non-adherent but the data shouldn't be lost. They just undergo another statistical model to account for their changes. Here is a nice video https://www.youtube.com/watch?v=Kps3VzbykFQ&t=7s +18  
dr.xx  Where does the question mention "intention-to-treat"? +  
notadoctor  They seem to be pretty obsessed with "intention-to-treat" it's been asked in one way or another in all the new NBMEs that I've done. (Haven't done 24 as yet) +8  
wutuwantbruv  They don't, intention-to-treat is just the best way to go about it @dr.xx +  
smc213  Great for ITT: https://www.youtube.com/watch?v=Kps3VzbykFQ +4  
yex  I agree with @notadoctor !! +  
ergogenic22  i think if it were per protocol, both groups would be excluded, the ones that were inconsistent, the ones that dropped out, and the ones that switched. But answer choices only allow ITT or exclusion of one group. +  


submitted by uslme123(57),
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sihT tqoisneu kamse no seens to .em ehS ash an exertemly wlo npnioge sprseeur tye hsa isngs fo nredcsaei aaacnirtriln prusre.es iDd etyh anem to upt 23 cm ???2???0H???

uslme123  Standard lab values are incorrect, way to go NBME. +3  
wutuwantbruv  I think they mean to put mm Hg. Normal CSF pressure is about 100-180 mm H20 which equates to about 8-15 mm Hg. +3  
alexb  I lost a bit of time wondering about that ugh lol +1  
mjmejora  I thought there must be an obstruction in the ventricles somewhere preventing csf from getting to the spine. so pressure is low in spinal tap but in the head it must be really high. +2  
donttrustmyanswers  Does anyone have clarification on this question? +  
llamastep1  Pseudo tumor cerebri can have normal ICP. Who knew +  
tyrionwill  Hi, mjmejora, MRI did not see anything abnormality, couldn't this mean that there was no obstruction in the ventricles? +1  


submitted by dr.xx(142),
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lCaoitngne daenlar yreipapalsh due ot -ely1xd2hsayor fcideyn.cie

t_kpp_okn1tw/_ir.ehypla_iea_asxah_d:i/eatgyu2eidd/eoiisgceeinoteilC/hcyiyarnndr.nolapf-rlswde

wutuwantbruv  Can't be 17α-hydroxylase because this would present with hypertension and some sort of ambiguous sexual presentation (males) or lack of secondary sexual development (females). Can't be 11β-hydroxylase because this would present with the opposite of the kid's presentation due to the production of 11-deoxycorticosterone (similar effects to aldosterone but not nearly as potent). The other two would not really make sense since there are increased levels of 17-hydroxyprogesterone. +  


submitted by gh889(115),
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hTe aesrwn is edu ot na pntcexoei uoietdln hree hewer anciin is dseu ni tsp /ow eaidtesb owh have teroacyrfr piryilegitmcryaehedr at ghih iksr ro hsa a hx of rnceapsita.it

I ergea thta tariebfs rea ftisr einl a(nd os oesd taht trail)ce btu NMEB swa ninhgo in on a cispcief cneieoxtp thta icanni cna losa be esdu cisen DLLV and TsG rae high ni mhiyr.pcrereidlgaeyit

heT e"ucl" yteh adh swa tcruerner" pce"tnsaairti whchi is depspuysol a dale atordsw anii.nc

I laso ptu srainece .D.L.H.

wutuwantbruv  Correct, you would not want to give fibrates to someone with recurrent pancreatitis since fibrates increase the risk of cholesterol gallstones due to inhibition of cholesterol 7α-hydroxylase. +  
kernicterusthefrog  FYI @gh889 can't follow your link w/o an NYIT username and password, unless there's a more tech-savvy way around that.. I appreciate the info, though. Niacin rx for familial hypertriglyceridemia w/ recurrent pancreatitis. Now I know.. +2  
impostersyndromel1000  Great points, very in depth knowledge taking place here. Also, familial hypertriglyceridemia (per FA 2019 pg 94) has hepatic overproduction of VLDL so picking this would have been the easiest answer (in retrospect) +2  
hyperfukus  @impostersyndrome1000 literally that's the ONE thing i remembered and i went YOLO lol cuz i was staring for a while +  
osler_weber_rendu  @gh889 I agree niacin is the answer, but even niacin causes increase in HDL. As if getting to the drug wasnt tough enough, NBME puts two of its actions in the options! What a shit question +1  
mtkilimanjaro  I forget where I saw (maybe UWorld), but I always thought increasing HDL is never really a primary form of lipid control. You want to lower the bad cholesterol etc. since increasing good cholesterol wont change LDL VLDL etc. +