There is a high indirect bilirubin concentration (8 mg/dl out of the total 10 mg/dl) so there must be a problem with conjugation. the enzyme needed for conjugation is UDP-gluronosyltransferase.

In this case, this is physiological neonatal jaundice. due to immature enzyme that resolves without treatment in 1-2 weeks. pg 387 FA 2019

I was between hepatic excretion of bilirubin and Hepatic UDP-gluco.... (Rotor syndrome and gilbert/physiologic neonatal jaundice respectively).

FA 387 (2019) helps where they say

• high UCB (indirect) --> Crigler Najjar or Gilbert syndrome, or physiologic neonatal jaundice.
• high CB (direct): think excretion defect for this type of vignette (Rotor Syndrome). Had the question said "with a direct component of 8" I think I would have chosen Hepatic Excretion of bilirubin.

Babies don't have mature activity of this enzyme when first born...Tx with phototherapy...not same mechanism as enzyme but makes bilirubin more soluble