They are asking, in a complicated way, the mode of action of Bethanechol. All the other options are not muscarinic receptors except parasympathetic stimulation.

This is the stupid way I've ever seen anyone ask a question

I kind of just thought about the muscarinic antagonist treatments for COPD exacerbation and asthma

• Tiotropium is a M3 antagonist used to treat COPD and asthma -> inhibits M3 -> airway dilation

• There fore if an M3 antagonist dilates airways than an agonist such as bethanecol should do the opposite -> airway constriction (AKA parasympathomimetic stimulation) it would sorta be like doing a methacoline challenge pretty much

FA 2019 p. 240

It says that in Cholinomimetic agents you should watch for exacerbation of COPD, asthma, and peptic ulcers in susceptible patients. I think this is what makes Bethanechol a contraindication. Bethanechol activates the bladder smooth muscle which would help urinary retention. I think this is what makes Bethanechol an indication.