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Retired NBME 25 Answers

nbme25/Block 2/Question#8 (reveal difficulty score)
The breakdown of dipeptides and tripeptides ...
Intestinal mucosa ๐Ÿ” / ๐Ÿ“บ / ๐ŸŒณ / ๐Ÿ“–
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submitted by โˆ—shak360(23)
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Dedeppitsi nda epriistdpte rea ucdeordp yb eht ivatcyit of sntpyri dan oytpcrhmsiyn hwinti eth eumnl of eth meou.dudn erFe naoim csadi aer uddcreop unpo ethrfur esntidogi fo thsee lsmla eppdtesi niihwt the hurbs redobr of teh intitnaels muaocs.

hTe lmenu fo hte demdounu is ownrg ecsbuae taht si rehew leragr peolmxc pstelpoyidep era kenorb dwon ot -id adn ipstitedper but teh aoticn of kiatgn -id dna rsdittpeipe wnod to iinuladvdi amoin dsiac phanesp ni hte eannsititl s.cmoua

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Maybe a way to remember this would be to think about Hartnup disease, an AR deficiency of neutral amino acid (primarily tryptophan is implicated) transporters in the proximal renal tubular cells and on ENTEROCYTES. This leads to neutral aminoaciduria and decreased absorption from the gut and little breakdown of dipeptides and tripeptides to amino acids in the intestinal mucosa. Decreased absorption from the gut causes decreased tryptophan for conversion to niacin and pellagra-like symptoms (diarrhea, dementia and hallucinations, and dermatitis in the C3-4 circumferential "broad collar" or "Casal necklace" dermatome, hyperpigmentation of sun-exposed limbs).

So if you can't convert dipeptides and tripeptides to amino acids in the ENTEROCYTES in the INTESTINAL MUCOSA, then you can get Hartnup disease.

+1/- shak360(23)

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