After the cuff is tied, the cells and tissue distal to the cuff will continue consuming ATP (ATP->ADP), but no fresh blood will be delivered to “clear” what will be an accumulating amount of ADP and other metabolites. ADP (=Adenosine) is itself a proxy of consumption and drives vasodilation of arteries! (Evolution is smart!) Increasing ADP/Adenosine in a “local environment” is a signal to the body that a lot of consumption is occurring there; thus, arteries and arterioles naturally dilate to increase blood flow rates and “sweep away” metabolic byproducts.
Which of the following reasons is why this question is bull?
1) Using the word "cyclic" instead of tricyclic for clarity
2) Knowing all of epidemiology of all drugs
3) having to reason out that anticholinergic effects are probably the worst over alpha1 or H1 effects to no certainty.
4) The crippling depression of studying for days-to-weeks on end to probably do average on the test.
If u are not goot in math like me xD. I did it this way: They gave me p^2=1/900 so p=1/30.
Now I need to find P, so use p+q=1. p= 1-(1/30)= 1-0.03= 0.97
So, 2pq= 2(0.97)(0.03)= 0.064 = (1/15) :D I got it right, this is my way to do it cause im not good at fractions.
You should be thinking of something like Enterococcus. They had a GU procedure and subsequent cardiac issues.
When I hear soft S1, i think that the patient must have had a issue with the closing of either the mitral or tricuspid valves. Playing odds, this should be the mitral valve. You also here an early diastolic murmur, so you might be thinking volume overload (S3).