to snoo-finity ... and beyond!
to snoo-finity ... and beyond!
After the cuff is tied, the cells and tissue distal to the cuff will continue consuming ATP (ATP->ADP), but no fresh blood will be delivered to “clear” what will be an accumulating amount of ADP and other metabolites. ADP (=Adenosine) is itself a proxy of consumption and drives vasodilation of arteries! (Evolution is smart!) Increasing ADP/Adenosine in a “local environment” is a signal to the body that a lot of consumption is occurring there; thus, arteries and arterioles naturally dilate to increase blood flow rates and “sweep away” metabolic byproducts.
Which of the following reasons is why this question is bull?
1) Using the word "cyclic" instead of tricyclic for clarity
2) Knowing all of epidemiology of all drugs
3) having to reason out that anticholinergic effects are probably the worst over alpha1 or H1 effects to no certainty.
4) The crippling depression of studying for days-to-weeks on end to probably do average on the test.
i thought in this way - all prostaglandins in this world help liquids to go ( eye aqua , afferent arteriole, etc) angina is due to ischemia , so something has to do with obstruction of fluids in this case blood , i exclude leukotrienes cause they has nothing to do with ischemia , and since its not leukotrienes only answer is TXA2
https://www.youtube.com/watch?v=i8OHZ8t6vg0 you guys WATCH THIS VIDEO
A related discussion (on a question from NBME 21) is here: https://nbmeanswers.com/exam/nbme21/714
can somebody explain how energy production by glycolysis increased, since aerobic glycolysis produce 32 net ATP,compare to 2 net ATP through anaerobic glycolysis ?
The person is presenting with symptoms of CHF (dyspnea on exertion, pitting edema) as well as b/l basilar crackles all of which indicate a fluid overloaded state. In addition, there is cardiac dysfunction with improper pumping of the blood. Due to decreased cardiac output, the body perceives this as a "hypovolemic" state and causes the release of hormones like ADH and Renin in an effort to reabsorb water to bring the volume up. While the patient is having extracellular fluid accumulation (edema) the arterial blood volume is low which is what causes the "hypovolemic" state.
rash seems to be that of shingles. Age range also matches up. Antiviral is the best option
I believe that metformin is contraindicated due to Patients with renal disease having a higher chance of lactic acidosis. This patient has increased BUN and Cr indicating renal disease
It appears that both the genitofemoral nerve (genital branch) and the ilioinguinal nerve both are external to the spermatic cord and at the superficial inquinal canal. Depending on location of the genitofermal nerve makes a difference for what it innervates. It appears that the genitofemoral nerve (genital branch) innervates the lateral anterior surface of the srotum, while the ilioinguinal nerve innervates the anterior surface of the scrotum.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5503924/
Google also says that the genital branch of the genitofemoral nerve can innervate the sensation of the skin of the anterior surface of the scrotum. Does it appears that either nerve choice would lead you to the correct answer?
Sources of nitrogen can be meat, eggs, and dairy product which he wasn't eating. The man had a low nitrogen intake but was still excreting nitrogen. Therefore, he had a negative nitrogen balance. A prolonged negative nitrogen balance may lead to a decrease in the plasma protein level, edema, anemia, lowered resistance to infection, increased susceptibility to certain toxic substances, the development of fatty liver or perhaps other serious sequel
Chronic Myelogenous Leukemia vs Leukemoid Reaction has a few differentiating features:
Basophilia can be seen in Myeloproliferative diseases such as CML. However, they are not seen in Leukemoid Reactions.
Leukocyte Alkaline Phosphatase (LAP) is often elevated in Leukemoid Reactions. But it will be decreased in CML because Abnormal cells don't make the normal enzyme.
Dohle Bodies are characteristic of Leukemoid Reactions. Not seen in CML.
As vshummy pointed out FA-2019 pg 424 has all this information as well as UWorld problem...I just can't seem to find the Q.ID. Maybe someone else can tag-team in.