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Welcome to match95โ€™s page.
Contributor score: 48


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 +17  (nbme18#32)
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sosnipartniTo fo het Avna eegn ofrm tianesto-ivsncrcnmya rcneutcsEoco si owh it afrrsenst .ticnssreae Tehy use otsnsaopnrs cihwh rae tcodlea no liadms.ps If you have dmaipls s,osl oyu to'wn eavh s,snatsrpoon nda ertnasisce lwli erdeeac.s

azibird  Why can't this be a point mutation? +4
freenbme23  I don't think that this implies that it can't be point mutation, but rather plasmid loss is more likely. Also, the point mutation Would have to ultimately lead to the plasmid loss. +

 +4  (nbme18#37)
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yuaUsll kbears wdon hoieyclu.ccnilns dauettM cueroeiehspnlesdstoa liwl otn baker nwdo ohccneliyculisn sa llwe &g-;-t inscredae lcyecnusnoihcil in ryuo myetss --t&;g nloreg ot overcer mrof teh atheet.csin


 +13  (nbme18#32)
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hiTs dlowu tjsu eb eht omarnl sseopern fo our seinkdy ot ewnh ew 'tdon indkr rtewa nhgeuo a(ak tusj ro.lna)m

mxloPrai uutlbe is tsincooi besaeuc ew ear nbsarbgrioe HBOT N+a dan O.H2

Mauacl denas is ctnopyoih euascbe ti is sgnisne hte dsalit cuonlevtdo eltuub -- eht somt LUITDE ptra fo hte phneron. bRrmmeee hte rtotrenuunercc nheecaxg ssteym - htkic aenidsgnc mbli is lnosig alCN nieu(r gtse essl tecanctdnreo as ti ca).ssedn

Mlardeluy oiclcgtnel cdtu si rciyoehnpt csbeeua ew era igenaosbrrb lla the aewtr kmagin het reiun oerm a.drcneotnetc


 +4  (nbme18#38)
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otneiusQ asw yckrit -- ndee ot kool tea TFEOLA v.eslle ohBt 12B ifindeycce dan ltfeoa yncdeeciif wlil acesu an esniearc ni nyteoiecho.sm nylO B12 eicdcfiyen llwi ecuas an craneesi ni TOHB iohmnecamyltl iadc DAN s.onytoehmice

cheesetouch  B12 deficiency 'BOTH be high' (and neuro sx) +

 +8  (nbme18#31)
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Ildaehn hncetastsei atth vahe wlo olsgoadb: tipaitron ciefnofbe/oiotlcd yuitlbilso dssvoeli in ldobo eomr iyas.el eref,hoTre heetr is SLSE gas nddeee ot usatrtea doblo -- kaa restfa taanuoistr fo d.oolb hisT eldsa ot rfseta iaausonrtt fo bnra.i

;tdlr wlo ibiuslltyo -g&;t- sftare osent -&t-;g ftresa ocrereyv

pelparente  great answer; just to add to it. Lipid solubility determines potency, not onset/offset (that is determined by blood solubility as stated above). The more lipid soluble the more potent the drug. The more lipid soluble the drug the higher the oil:gas partition (directly proportional to potency) and the lower the MAC (inversely proportional to potency). Source: Boards and Beyond General Anesthesia Neurology +2
cbreland  I really went down a rabbit hole on this one... Convinced myself that it had something to do with mask v. IV anesthesia even though I haven't seen/heard that anywhere. Taking step in a week๐Ÿ™๐Ÿผ +
jer040512  I thought a low blood:gas partition coefficient meant that it has a low solubility and therefore DOESN'T dissolve in the blood that easily. +2
dhpainte22  Think onset/offset is about blood solubility and potency of drug higher with higher lipid solubility so low blood gas coefficient has faster onset and recovery. +
jurrutia  Just to drill point home further: consider halothane, high lipid solubility (so high potency) and high blood solubility (slow onset). If you want fast onset, low solubility is the key. +

 +2  (nbme18#13)
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mnmuIe ymtehnbocroitoap F(A 207,1 gp )504 - sucesa odrtecntusi fo l-baetytlnpaotdei pecmlxo yb selnep enladig ot edrcaese in epettlla ANYTIUTQ nad crniedaes smcaaeeokytgry no beon orwamr io.ybsp

ellie0124  anti-GpIIb/IIIa antibodies +
baja_blast  FA 2019 p. 419 +
jurrutia  May be secondary to viral illness... +




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