Anybody have any good insights as to what is going on here? Does surgery somehow cause hypokalemia? Or does this have to do with digoxin toxicity? I'm not sure how surgery fits in. Thanks in advance!
My two cents: Abdominal surgery for perforated gastric ulcer probably resulted in hypovolemia, leading to increased RAAS activity and contraction alkalosis. This leads to hypokalemia, which is a factor that predisposes to digoxin toxicity. PVCs are one of the most common digoxin-associated arrhythmias and are often the first sign of toxicity, completing the clinical picture described in this question.