My understanding is that H. pylori actually decreases the risk for esophageal disorders (ex. adenocarcinoma)
How would a chronic H. pylori infection lead to healing ulcer/ stricture formation or adenocarcinoma?
GERD --> acid damages mucosa --> knockout of the mucosa leading to decreased stem cells --> ulcers form and heal via fibrosis --> stricture --> dysphagia. Complication of this chronically would be Barrett Esophagus? (I can't tell if those are cells filled with fat or goblet cells that replaced the squamous epithelium). Barrett Esophagus can progress to Adenocarcinoma of the esophagus. Source: Pathoma pg 102
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submitted by ergogenic22(321), 2019-08-11T23:38:16Z
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