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Retired NBME 20 Answers

nbme20/Block 3/Question#15 (reveal difficulty score)
A 23-year-old woman, who was diagnosed with ...
Substance P ๐Ÿ” / ๐Ÿ“บ / ๐ŸŒณ / ๐Ÿ“–
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submitted by โˆ—poisonivy(39)
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Per UWorld; capsaicin found in chili peppers causes excessive activation of TRPV1 (transmembrane channel), which increases intracellular calcium that results in long-lasting dysfunction of nociceptive nerve fibers (defunctionalization). It also causes release and depletion of Substance P, causing at first burning and erythema but over time causes decreased pain sensation.

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submitted by โˆ—hayayah(1212)
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Substance P (SP) is an undecapeptide present in the CNS and the peripheral nervous system. A compound thought to be involved in the synaptic transmission of pain and other nerve impulses.

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mambaforstep  also, apparently enkephalins attenuate substance p... so it would be dec her pain. +1



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submitted by โˆ—step1soon(51)
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Capsaicin induces the release of substance P, one of the principal chemo-mediators of pain impulses from the periphery to the central nervous system (CNS). After repeated application, capsaicin depletes the neuron of substance P and prevents its reaccumulation.

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submitted by โˆ—ssc30(3)
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Per FA2020 pg 551.

Substance P release is inhibited with opioid analgesics. Therefor connecting the relationship between Substance P and pain. Enkephalin is a type of opioid receptor which is involved in this pathway but is not a neurotransmitter as the question asked for.

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submitted by kp36(1)
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Capsaicin can also reverse mucosal damage caused by ulcers

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 -1  upvote downvote
submitted by โˆ—poisonivy(39)
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Per UWorld; capsaicin found in chili peppers causes excessive activation of TRPV1 (transmembrane channel), which increases intracellular calcium that results in long-lasting dysfunction of nociceptive nerve fibers (defunctionalization). It also causes release and depletion of Substance P, causing at first burning and erythema but over time causes decreased pain sensation.

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chandlerbas  also topical Rx for post herpetic neuralgia +1



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