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bingcentipede
I think it's because of the last sentence - asks about a defect in an inhibitory neurotransmitter, with glycine being the only possibility. I think it's one of those "here's a stem, but just look at the last sentence" questions.
+6
cassdawg
This actually is not tetanus or botulism. The deficit has been present since birth. He has glycine encephalopathy, a rare disorder (https://en.wikipedia.org/wiki/Glycine_encephalopathy). Definitely could be tricked into thinking botulism but defect in "inhibitory neurotransmitter" points to glycine deficit as glycine is the only inhibitory neurotransmitter listed!
+4
cassdawg
(Disclaimer that I am assuming his deficit is just a weird kind of glycine encephalopathy because normally its a disease of metabolism not receptor; but it presents with the hiccups and seizures like seen in this baby. Big thing is the last sentence as was already said)
+2
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submitted by โcheesetouch(250)
Glycine is the only inhibitory neurotransmitter present. He likely has glycine encephalopathy is a rare autosomal recessive neurological disorder. Tetanus toxin - blocks release of inhibitory NT's gaba and glycine. Botulism - 'floppy baby ate honey' - inhibition of ACh release at the neuromuscular junction.