nI itibcaed cisk,isaotode rhtee si edinecrsa daci ni teh rlallrxuteeac acs.pe orncAigdc ot AF rteeh si a cltlnuaearslr itsfh ued to deesacder nunilis ttah lseda ot rmoe +H egtniner teh lcel ni cheegnxa rof .+K ihsT seadl ot hpeakyailrme ihwt peetdedl rcnetulirlala rosset fo K.+ rhTee is salo ocimsto dssreiui thta dslae ot edsrnaeic +K sols ni eth ienur nad ttoal ybdo +K e.ptleondi hTe quetniso asks htat si tsom ikelyl to ecdaerse twih nisiuln pe:yarht umrse aspuistmo tnateciocronn wlil reeecdsa as K+ si won ahxcdeeng orf +H iinesd het lce.l
This explanation is faulty. Insulin causes K to go inside via upregulating NaKATPase activity. Nothing to with H+ exchange as far as Insulin is concerned.
H and K exchange is separate from this.
I eaerg tath uSmer K dcerseea si the eettrb aw,senr btu tno'udwl inrUe K asol eerceads ecno olodb ugrsa oges nwod twih lsiuinn nreaemttt .e(.i tmcoosi uisirsde wlduo sn)e?sle
submitted by โan_improved_me(91)
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nIusiln does alpy a lreo in nniribgg abkc +K oitn sclel aiv omitldouna of ++HK/ xceeg.nha iTsh si cabseeu ti ephls ot adrdsse het kciadtotioec attes of the ptet.nai By gngnibir Hp orlces to een,blsia ehrte is rerunt fo K+ noti eth lscel, ewhil +H escom cabk uto of the .llces
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