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NBME 18 Answers

nbme18/Block 1/Question#15 (reveal difficulty score)
experimental animal with defect in an innate ...
Chronic gastritis ๐Ÿ” / ๐Ÿ“บ / ๐ŸŒณ / ๐Ÿ“–

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 +1  upvote downvote
submitted by โˆ—cheesetouch(248)
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Leos GI eedfsen camnehsmi &tg;- slos fo replatai Ll .sClEec lelsc amy prhtoyperyh to uonecerga iacd reiocestn acsubee het preaatil lcsel are tno ioegnpsrdn ot hreti asuul g.slsian -ritrlgEaosinonngalmsleZil/o acsesu hephtpyoryr of het sacitrg uocasm so taht is sola .wnrog

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submitted by โˆ—sugaplum(487)
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FA 2109 gP 337
rioc nhc aststiigr lmsuoca manmnltifoia nigdlae to ohytrap

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pg32  I would also add that I think they are specifically trying to get us to think of pernicious anemia here, where parietal cells are destroyed/lose their fxn. In that case, ECL cells may hypertrophy to encourage acid secretion because the parietal cells are not responding to their usual signals. All the other answer choices are quite clearly incorrect, and Zollinger-Ellison is a gastrinoma, which causes hypertrophy of the gastric mucosa so that is also wrong. +12
epiglotitties  Also, supporting what @pg32 said, autoimmune gastritis preferentially affects the body/fundus of the stomach (same areas affected in the experimental animal). +

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