taentiP mya vhea edhtriraye md,iaaneoge hwich is issaetaocd ihwt eurct"renr tastcka of tinen,es vsmsae,i iczedoall cstausoueunb eeadm onivgvlni het ret,simietex nae,igliat fa,ec ro nk,tur ro ouucmsbsal emdea fo pperu awaryi or l"sebow. heT talrcie egos on ot sya -a1s"reetsCe hiniirotb roksw cryeidtl no eth eotemmpcnl dna ocntcta amslpa csesacda ot uceder riibkndany rsaeeel" cihhw si salo yplrbabo good to wn.ok
Hereditary angioedema (HAE) caused by C1-esterase inhibitor deficiency is an autosomal-dominant disease resulting from a mutation in the C1-inhibitor gene. HAE is characterized by recurrent attacks of intense, massive, localized subcutaneous edema involving the extremities, genitalia, face, or trunk, or submucosal edema of upper airway or bowels. Routine long-term prophylaxis with either attenuated androgens or C1-esterase inhibitor has been shown to reduce the frequency and severity of HAE attacks.
This is what I came across putting the two (angioedema + laryngospasm) together. Take into account that this info is regarding ACEIs but, I guess with a certain severity, angioedema can be associated with other sx. "However, angioedema has occurred suddenly after months to years of therapy, and about 20% of known cases of angioedema occurring in this context may involve severe symptoms (e.g., dyspnea, stridor, laryngospasm)."