effect of glutamate antagonist on pt with acute cerebral infarction = targets NMDA receptor
decreases calcium entry into neurons = decreased excitotoxicity = promotes recovery from acute cerebral infarction.
Tihs noe aws ticrky tbu I thnik uyo odeclโuv oedn hits noe wttoihu oenedlgwk fo DNAM proetrc.se Setm dtlo yuo atth ameatglut istacveat tohb DMnAnoN- dna MADN ecroesptr tub ti eavtdciat ynol NMnDo-nA ceerosrpt ni hte yealr esha.p hTat asnem DMAN porreestc ticaaevt etafr -MNDnoAn tpe.ocerrs That nsame ntmeihosg wsa dgaiyenl DAMN etoreprc vttncgaaii nda hte ylno raewsn thta dame nssee as het Mg hningiiitb MNDA at gnisetr e.ttapniol Ocne teh clle is rleeopzaddi by MNonn-DA pesrr,tceo DANM operertcs anc be atca.eidvt
tA girenst mnbearme lettniapo het DNMA tescorrpe ear cdeklob by gM.2+ Teh gavleot enetendpd gM+2 bokcl si drveelie pnou oepzriildoatna of eht asp-cntyipsot ernaemmb . Teh igadnl epndtdene nvcaiottai fo the DNMA reeptcor sreqeriu niao-tovaictc yb two isdn,gal maylen guelmtata and genl.icy
nA atomnritp rdlteea aicilnlc pinicpolata is eeernrmmg:bi avirnOa aametort nda reiht nacssioioats wtih: -tDinNMAA eoterrcp hei.ascielntp sihT pertsnes s:a chasiriycPt aibu,desncrt yrmmeo dsitfi,ce eus,reisz e,sasniyisdk ainctmuoo ilti,atnsbiy lugegaan dfincosnuy.t
:FA 0;202 pg 228
ont'd tegorf oabtu the gM lbco!k . . . . . .
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