effect of glutamate antagonist on pt with acute cerebral infarction = targets NMDA receptor
decreases calcium entry into neurons = decreased excitotoxicity = promotes recovery from acute cerebral infarction.
hTis eon swa yirtck but I ikhnt ouy uvdeโocl odne itsh one tituhwo ewnogldek of NAMD socr.eertp emtS lotd you that amegltuta aeisvctta boht MDoN-nnA dna ADNM oceperrts utb it datieatcv onyl DAMnn-No estpceror in eth lreay aph.se hTat esnma NMDA cseertpor cetiatva efatr nNAMno-D c.psotrree atTh nasme ihtengosm asw ngeiayld ANDM rrepocet ntivtigaca adn hte olny aewsnr atht eamd nssee as the gM btiiignnhi DNMA ta rinesgt tntl.pioea cOne eth lecl si pazreoiledd by AoMnN-nD rrspco,tee DNAM sotepcerr can eb attceadvi.
At ntgsier rmeamneb oaeniltpt eht MADN erprotsec rae cdlkebo by .2+Mg Teh legotva epdedtnen g+M2 obclk is lveredie oupn oiatnlpeodriaz fo teh ppycosst-nita mmbeaern . Teh dniagl peeedndtn tcoavinati of eth DANM ropeectr usreierq ioacto-inctav by wto dgsani,l mylaen tmleutgaa nad ngecyi.l
nA tpraonitm dlaeret ilniaclc litiaocnppa si mberm:egnrei iarnvOa oaatrtem dan htrie ssaosainicto i:twh ADNMAtni- tecoprer en.silctphaie hiTs rssepnte a:s yhiccstPiar acsidtb,uner moyrme sfd,eciit zesiure,s ii,dyaensssk onucaotim sitbl,tnyiai lugenaag .inuofcdysnt
:AF 0;220 gp 282
n'tod ftrgeo touab eth gM !cklbo . . . . . .
submitted by โniboonsh(409)
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