Wikipedia pathophysiology section of "retinopathy of prematurity" explains this pretty well. Retinal vessel maturity is incomplete in premature infants--the process of continuing this maturation in these babies is directed towards growth of vessels in O2 deprived areas, but if O2 is being supplemented, this process is hindered. Therefore when O2 supplementation is removed, excessive vessel proliferation going into the vitreous humor (not where it is supposed to be) leads to retinopathy of prematurity (defined as "disorganized growth of retinal blood vessels leading to scarring and retinal detachment")
r"ohncic ghih xongey stinrosatua acn daevreyls etfcaf ngul nad eye tuemocso fo mrrptee isn"a t.fnnlik
eraeg htiw '. btuerehrkay ygrnit to yas atth ryaiprm oagl fo teeanmttr wthi neslpmuaeltp 2o is to aemk esru eehtr is on nlireat dg.amea
why is protocteni fo teh cdrioho supl,ex mrof vrtleiainucantrr truruep orasdncey to eaignmrl mxitra oareghrmeh ton a piseolsb sera?nw
submitted by โhyoscyamine(59)
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