Wikipedia pathophysiology section of "retinopathy of prematurity" explains this pretty well. Retinal vessel maturity is incomplete in premature infants--the process of continuing this maturation in these babies is directed towards growth of vessels in O2 deprived areas, but if O2 is being supplemented, this process is hindered. Therefore when O2 supplementation is removed, excessive vessel proliferation going into the vitreous humor (not where it is supposed to be) leads to retinopathy of prematurity (defined as "disorganized growth of retinal blood vessels leading to scarring and retinal detachment")
croc"nih hhig yoexgn ustaioantsr cna vysalrede acteff nulg adn eye oeomscut fo tmrepre t"nf as.niilnk
regae htwi y b'uehtkaer.r rygitn ot say atth pyimrra oagl fo eamnttter tiwh lptslmepaeun o2 is to ekma sure trhee is on raenitl .amgdae
why si nroetpciot fo hte odhicro p,leuxs ormf eitrrtncilvaanur rpuretu aoncedrsy ot meaniglr miaxrt gerehhmoar nto a pslsobie wrasen?
submitted by โhyoscyamine(59)
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