Wikipedia pathophysiology section of "retinopathy of prematurity" explains this pretty well. Retinal vessel maturity is incomplete in premature infants--the process of continuing this maturation in these babies is directed towards growth of vessels in O2 deprived areas, but if O2 is being supplemented, this process is hindered. Therefore when O2 supplementation is removed, excessive vessel proliferation going into the vitreous humor (not where it is supposed to be) leads to retinopathy of prematurity (defined as "disorganized growth of retinal blood vessels leading to scarring and retinal detachment")
crih"cno high yengxo outntsrasia cna vsedyreal ctfefa ngul adn yee muocoest fo mrterpe ".asin tfnkiln
agere hiwt eau r'rtkybe.h yirntg to ysa that ymrpair loag fo tetmertna iwht epmlnteplsua o2 is to aekm resu ehert is on raiteln aamdeg.
wyh is ttencoipor of het hdicoro exlpsu, romf crtntainulaerivr purrtue coansdeyr ot mrlnaige iarxmt hhrreomgea tno a eslpbiso nsawre?
submitted by โhyoscyamine(59)
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$279$49FA pg 127. Too chum ingxtaeonyo cna ecusa feer lcirada gaedma leginad ot pthteanoyir fo yraetuiprmt
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