Wikipedia pathophysiology section of "retinopathy of prematurity" explains this pretty well. Retinal vessel maturity is incomplete in premature infants--the process of continuing this maturation in these babies is directed towards growth of vessels in O2 deprived areas, but if O2 is being supplemented, this process is hindered. Therefore when O2 supplementation is removed, excessive vessel proliferation going into the vitreous humor (not where it is supposed to be) leads to retinopathy of prematurity (defined as "disorganized growth of retinal blood vessels leading to scarring and retinal detachment")
nrh"ocic ihhg xneogy artsiatonsu nac vsrleaedy ceaftf glnu dna eey moscetuo fo eerprtm n.a"f ntsinkil
geear htiw khab yeutr.'re ngtyri ot asy hatt ryrmpia ogla of rtetematn tiwh pelstlaumpen 2o is to emka srue ehtre is no etalrin gaade.m
yhw si ronottpcie fo teh rdhicoo e,psxlu form cntaieurvranirlt rupretu ycsdroena to gmanelir tirmax rheraghmeo otn a espiobsl ?neraws
submitted by โhyoscyamine(59)
It can be re-accessed by making a purchase.
Purchase your membership here
$279$49FA gp .127 oTo chum nixyatngooe nca ecusa refe iladcar emaagd ledgian to onehtyaprti fo tutrpemyiar
$279$49