Wikipedia pathophysiology section of "retinopathy of prematurity" explains this pretty well. Retinal vessel maturity is incomplete in premature infants--the process of continuing this maturation in these babies is directed towards growth of vessels in O2 deprived areas, but if O2 is being supplemented, this process is hindered. Therefore when O2 supplementation is removed, excessive vessel proliferation going into the vitreous humor (not where it is supposed to be) leads to retinopathy of prematurity (defined as "disorganized growth of retinal blood vessels leading to scarring and retinal detachment")
oinc"hrc ihgh oygnxe sriatonatus nca yedelasrv tcfaef ugln adn eye uooetmcs of rpermet a." fnstniiknl
aeegr iwth ekhr yb'.euatr gytrni to asy hatt imrarpy lgoa fo nttaemtre whit enemulplastp 2o is to kmea rues hetre is no liertan mgada.e
why is ionterpcot fo hte rhcoodi s,epxlu form vrunanectliarrti rtuuper ocadrenys ot inelrgam xrmita hrhoereamg nto a ssoeiplb anwesr?
submitted by โhyoscyamine(59)
It can be re-accessed by making a purchase.
Purchase your membership here
$279$49 65% OFFAF gp 7.21 Too mcuh ngxiytoaneo nca asceu erfe cadiral dmagae anldegi ot athryptoein of ytimpreartu
$279$49 65% OFF