nehW in tbodu sit oblyrpab het ieclSnp ineV lyap(" ds"do - nGja)ol
Jsut a narodm ftcaido (as arf sa I n,ok)w ni tesptani twhi ctiisaetpnra eht stom lilyke vlesse ofr boihosmsrt is het snpceil ievn ued ot sceol moia“nact ste”i iwth eth ar.npscea Tihs wlduo aslo ucsae prticso-ngeasl sevia,rc nepialginx the miigvont of odolb.
tMso mmoonc ceuas of sencipl vine htssboomir si ircochn cpiaaenirstt, ucdsea yb ueepsrivno namonaimlft.i
UW: the short gastric vv drain blood from the gastric funds into the splenic vein, pancreatic inflammation (e.g. pancreatitis, pancreatic ca.) can cause a blood clot w/i the splenic vein, which can increase pressure in the short gastric veins and lead to gastric varies only in the funds
Chronic alcohol-related hematemesis is associated with 2 possible sources: gastric varices or esophageal varices.
Gastric varices -> L. gastric vein or R. gastric vein (draining the gastric fundus into splenic vein)
Esophageal varices -> L. gastric vein
L. gastric vein can be ruled out because splenomegaly wouldn't be present in a L. gastric vein thrombosis, making the splenic vein the correct answer.
Isolated gastric fundal varices are seen in splenic vein thrombosis.
Thrombosis increases pressure in the short gastric veins --> gastric varices only in the fundus