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nbme24/Block 4/Question#17 (52.6 difficulty score)
A 63-year-old woman undergoes operative ...
Decreased movement through the arachnoid villiπŸ”,πŸ“Ί
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 +14 
submitted by happysingh(45),
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so, teh eKy sordw tath no oen si ignimnnteo : itcmcmiunonag olapuhrscydeh

teh yaptopshh ogse ielk siht :

na ymaomnatirlf inetgst .(i,.e ndohbsiucara gma)rhreeho yldie fsrobisi / ncrsagri of the cdhnroiaa oaiuntglnsar g=;t& idaermip CSF rinaadeg

hte eyk itpnos / opscetnc ythe rea rtiygn to ttes rehe : 1. do oyu konw thwa mtnccniaoguim hochpyuesrald (hwtuoti emth eltlngi you hoset )dsowr .2 od oyu nwko h'wats het ohypshogptyaoli of( niiumatcncmog esdoclha)puhry si ?

potentialdoctor1  Exactly. To add to this, communicating hydrocephalus can be subdivided as follows: Normal-pressure hydrocephalus: Chronic/gradual decrease in CSF reabsorption at arachnoid granulations, usually due to calcification due to aging. CSF accumulates slowly, so ventricles are able to widen without causing an important increase in intracranial pressure. Symptoms occur due to compression of periventricular white matter tracts ---> Wacky, wobbly, wet High-pressure hydrocephalus: Acute decrease in CSF reabsorption at arachnoid granulations, usually due to inflammatory state in the subarachnoid space (eg, meningitis, sub-arachnoid hemorrhage). CSF accumulates suddenly, causing an acute-onset increase in intracranial pressure +7  
sunshinesweetheart  not to take away from your perfect explanations, but if it were a woman with neck stiffness and fever (rather than circle of willis rupture) that could lead to increased CSF production, right? I think that's the only case where CSF production would increase. Also I think decr absorption in arachnoid granulations in that situation as well so it'd be a shit question +  
peqmd  If anyone like me also got "decreased absorption in choroid plexus", as their wrong answer it's because the choroid plexus doesn't "absorb" it produces. +9  
alienfever  FA 19 p510 +2  
alienfever  If anyone chose F, communication hydrocephalus is caused by decreased absorption and not increased production. FA 19 p510. +1  
an_improved_me  So she has a leaking aneurysm for how long.. gets it repaired, and then within 2 days has an inflammatory response that leads to decreased CSF absorption at arachnoid granulation... Is it the bleeding associated with the aneurysm causing it? The surgery? I'm inclined to say the latter, given that it happens coincidently after the surgery, and not for however long it was leaking beforehand. Thats what tripping me up. +  



 +4 
submitted by colonelred_(107),
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rNllayom eth aniahcord lilvi nrdsia eht FCS ofmr the nhrdsaauboic caeps to teh uvnoes esst;ym fi hist ratp csmoebe fevcetied enth oyu nca magieni lal ttah CFS now iguldbni up ni hte unhrdacbaois epas.c

keycompany  Also take into account this patient had surgery that requires penetration into the subarachnoid space (hence through the arachnoid mater). This can lead to scarring of the arachnoid granulations and subsequent communicating hydrocephalus. +16  



 +2 
submitted by roygbiv(20),
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Teh pitntae hsa a aigkenl bryer mnayurse gt;&-- this sdael to a iuhsbcnaaord hrogemaehr ro( lkeeaga onit iscaoahnrbdu csp)ae &-g;t- uceta HAS nca elda ot rdecesade ntsobaopri and movetnem avi ochinraad livil

zbird  So this patient has a leaking berry aneurysm which undergone to surgery and repaired but in two days she developed widening of the SA space which could be explained by increased CSF production but a decreased in absorption, which is due to blockage of the arachinoid granulations by the leaked red and white cells therefore there is a decreased movement of the CSF via the arachinoid villi +1  



 +2 
submitted by diabetes(28),
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iymlps eth oodlb dseiin teh SFC bceemo lokcdbe tohughr nadacriho goasninatrul =;tg&= edasecer otipbornas fo nnCu&;icgFSaco==mgttim loprsyuacd.heh




 +1 
submitted by krewfoo99(93),
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Wyh ludow dcseaeedr mmveetno htrhgou teh crbleeaer uuatqcad be ong?rw tWih lal teh uidbl fo olobd ni eht SFC tartc tuohtiw rpnotsa,oib wntldou oetvenmm alos eb dcaeesred tgorhhu eth a?udectuq

ergogenic22  this would cause a non-communicating hydrocephalus with enlarging of the lateral and 3rd ventricles but normal 4th ventricle and subarachnoid space +1