oS the estb i odluc ifdn swa in iFsrt Adi 0291 gp 634 edrun iabeiDtc to.dcKseasoii Teh ygieclpayhmer dan lieahekrpyma ascue na osocimt isuedrsi so hte erinte doby stge petledde fo d.ilfsu eencH ywh rpta fo eth tatetnemr for DAK si VI u.ifdls You mihgt neve eryl no ttha cpeei fo fnrioimanto anloe ot neswar htsi eqi,sotnu taht AKD is taedert tihw VI i.dlfus
syas atht rthoysyamliepor nda asdcisio rltae nlatme au.sstt
ihhcw ltraeoecrs twhi hte hgrti sawren
p.s i ogt ti wrgno too :)
Idk how you could say that it's from extracellular dehydration, but whatever I guess.
this is how I looked at it extra cellular osmoles> intracellular so it will pull the h20 out.... then the high osmotic pull of the sugar overwhelming the SGL2 transporter in the kidney will pull the h20 out of the body dehydrating the extracellular compartment
a little messed up, but "Inability of neurons to perform glycolysis" seems like a tempting answer. But the reality is, the neurons are able to perform glycolysis, they ready to rock but just waiting on insulin. I still chose this as my answer tho.
I guess this is one of those choose the best answer questions. I think FA should add the reasoning behind cerebral edema, being that it's a major cause of death (but I couldn't find it in Robbins either). Having so much glucose in the blood vessels causes water to be drawn out (ICF --> ECF). So that's a intracellular dehydration.